This paper proposes a new methodology to better assess indoor RF-EMF exposure in buildings near telecommunication base station antennas by refining measurement-point selection. Implemented in four multi-storey buildings in Natal, Brazil, indoor electric field peaks and averages were reported to be substantially higher than ground-level measurements. Although the highest indoor levels remained below ICNIRP recommended limits, the authors argue current regulatory evaluation methods may underestimate indoor exposure in certain building locations.

This exposure assessment measured RF power density across different floors of a high-rise university building using a spectrum analyser and log-periodic antenna. Power density decreased from the ground to the third floor but peaked on the fourth (top) floor. The 900 MHz band showed the highest reported power density (1.16E-03 W/m2), and the authors suggest higher-floor occupants may experience higher RF exposure from nearby communication towers.

This exposure assessment measured personal RF electric field strength in multiple indoor and outdoor micro-environments in Malaysia using an ExpoM-RF 4 meter and modeled exposure with machine learning (FCNN, XG Boost) and linear regression. Reported exposures were usually below the stated public limit (61.4 V/m), but maximum values in dense urban areas with many base stations were reported to approach 56.7365 V/m. The authors frame near-threshold maxima in high-density areas as a potential health risk and recommend caution and monitoring.

This exposure-assessment study uses FDTD simulations to evaluate auto-induced downlink RF-EMF exposure at 3.5 GHz when downlink energy is focused toward user equipment near the head. Exposure varied substantially by device position (ear, eyes, nose) and by the precoding technique used. The authors report that the choice of normalization strategy can produce cases where ICNIRP basic restrictions are exceeded even when reference levels appear compliant, motivating a precautionary framing for compliance assessment.

This rat study examined repeated 3.5 GHz RF exposure (2 hours/day, 5 days/week for 30 days) and thyroid-related outcomes, with and without quercetin. The abstract reports altered thyroid hormones (lower T3/T4, higher TSH) and increased oxidative stress in thyroid tissue after RF exposure. Quercetin appeared partially protective, though effects were not uniformly statistically significant, and SAR simulations indicated relatively higher absorption in the thyroid region.

This narrative review discusses low-intensity RF-EMF exposure, primarily from mobile phones, with a focus on thermoregulation and energy homeostasis. It reports that many rodent studies at 900 MHz describe cold-like thermoregulatory and behavioral responses and molecular findings suggestive of WAT browning, while BAT transcriptional changes typical of cold exposure were not observed. The authors indicate short-term adaptations may not disrupt homeostasis, but emphasize uncertainty about long-term consequences and call for further research, including at 5G-relevant frequencies.

This animal study measured core body temperature in free-moving male and female Sprague Dawley rats during and after 3-hour exposure to 1.95 GHz RF-EMF at multiple whole-body average SAR levels. A measurable thermal response was reported at 4 W/kg, while lower SAR conditions showed smaller or no significant temperature increases. The authors note that temperature dropped quickly after exposure ended, implying post-exposure measurements may underestimate peak heating.

This laboratory study examined whether a 900 MHz radiofrequency electromagnetic field (RF-EMF) influences the behavior of newly emerged honey bee workers. Compared with controls, the exposed groups showed behavioral changes, with some parameters showing statistically significant differences seven days after exposure. The authors frame RF-EMF as a potential environmental stressor and call for further research, including gene expression analyses.

This paper is a commentary reviewing how Bradford Hill’s epidemiological criteria can be applied to multidisciplinary evidence on RF-EMF exposure and adverse health effects. It reports that systematic reviews and meta-analyses in this area often reach substantially different conclusions, and argues that key weaknesses in primary studies—especially exposure measurement error and insufficient time for long-latency tumors—help explain the divergence. The author suggests these limitations may cause underestimation of potential causation if the associations are truly causal, and calls for independent guidelines to improve future epidemiological research quality.

This review discusses potential impacts of radiofrequency electromagnetic fields from technologies such as Wi‑Fi and mobile phones on the central nervous system, neurotransmitter dynamics, and reproductive health. It describes proposed mechanisms including oxidative stress, thermal effects, altered neurotransmitter activity, ion channel changes, and neuronal apoptosis, while acknowledging conflicting evidence. The authors note that Wi‑Fi RF exposure has not been confirmed to exceed safety guidelines but argue that updated standards and long-term studies are needed, particularly for children/adolescents and in the context of expanding technologies such as 5G.

In a rat experiment (n=59), a single 40-minute whole-body 28 GHz exposure at near-threshold WBA-SAR levels was evaluated under normal and heat conditions with restraint. After accounting for sham-related restraint stress, exposure was associated with increased serum-free corticosterone 1–3 days later, especially when rectal temperature rose by >1°C. Urinary catecholamines suggested an immediate inhibitory effect on stress response (notably noradrenaline), with heat amplifying effects and linking noradrenaline to tail surface temperature.

This animal study reports that prolonged RF-EMR exposure (2.45 GHz for 8 weeks) increased oxidative stress and ferroptosis in mouse testicular tissue and was associated with reduced sperm quality. Melatonin administration reportedly mitigated oxidative injury and inhibited ferroptosis. The abstract attributes the protective effect to Nrf2 pathway activation via MT1/MT2 receptors.

This systematic review evaluated RF EMF exposure and cancer outcomes in experimental animals, including chronic cancer bioassays and tumor-promotion designs. Across 52 included studies, the authors report high certainty of evidence for increased malignant heart schwannomas and gliomas in male rats, and moderate certainty for increased risks of several other tumor types. Many other organ systems showed no or minimal evidence of carcinogenic effects, and the authors note challenges in translating animal findings to human risk assessment due to exposure and mechanistic uncertainties.

This paper presents a mixed-method study protocol examining electromagnetic hypersensitivity (EHS) in relation to mobile phone radiofrequency radiation exposure among undergraduate students. The quantitative component aims to identify predictors of EHS using a biopsychosocial model, while the qualitative component explores individual experiences through in-depth interviews. The abstract provides study design details and sample size but does not report study results.

This exposure assessment measured outdoor RF radiation from 4G/5G base stations near 10 schools in Örebro, Sweden (October 2024). The authors report maximum levels of 10,716–68,452 μW/m² and state these are far above EUROPAEM EMF guideline ranges; two locations also showed higher peak readings with a second meter. The paper frames these findings as indicating a significant health risk, particularly for children and sensitive groups, though no health outcomes were measured in this study.

This animal study exposed male Wistar rats to 900 MHz RF-EMR from a mobile phone for 1 hour daily over four weeks and assessed behavior and tissue changes. The authors report altered contextual fear conditioning-related behavior in exposed rats. Histological assessments indicated apoptosis and enlarged perivascular space in the hippocampal CA3 region and apoptotic/inflammatory-like changes in the adrenal zona fasciculata, with no reported differences in adrenal medulla cytoarchitecture.