This PubMed-listed in vitro study tested whether 1800 MHz RF-EMF exposure can modify chemically induced DNA damage in mouse embryonic fibroblasts under standardized, non-thermal conditions. The authors report RF-EMF alone did not produce detectable DNA damage and did not significantly increase damage from hydrogen peroxide, 4-nitroquinoline-1-oxide, or cadmium. However, co-exposure with hexavalent chromium (Cr(VI)) was reported to synergistically increase DNA damage in the comet assay, which the authors interpret as possible selective exacerbation of Cr(VI)-induced genotoxicity requiring further investigation.

This in vitro study tested whether 1800 MHz RF-EMF modifies chemically induced DNA damage in mouse embryonic fibroblasts under non-thermal exposure conditions. RF-EMF alone did not produce detectable DNA damage and did not significantly enhance damage from hydrogen peroxide, 4NQO, or cadmium. In contrast, co-exposure with hexavalent chromium (Cr(VI)) was reported to synergistically increase DNA damage, suggesting a selective co-genotoxic interaction under specific chemical conditions.

An RF Safe commentary argues that the U.S. Department of Health and Human Services (HHS) is violating Public Law 90-602 by failing to continuously update radiation-safety standards, asserting that no formal revisions have occurred since the mid-1980s. The post links this alleged inaction to continued public exposure from wireless technologies and calls for renewed long-term research and stricter exposure limits. It also claims the National Toxicology Program (NTP) was shut down in 2024 and references a 2021 court decision criticizing FCC RF rules, urging congressional action and new legislation.

This rat study tested whether 2600 MHz radiofrequency field exposure interacts with indomethacin to affect gastric tissue. Both exposures alone were reported to increase oxidative stress and reduce antioxidant markers in the stomach. Co-exposure was reported to intensify oxidative stress, apoptosis, and histological gastric mucosal injury compared with either factor alone, consistent with a synergistic detrimental effect in this model.

This laboratory study tested combined effects of constant temperature and RF exposure on development of Aedes aegypti and Aedes albopictus from hatching to adult emergence. Temperature was reported as the primary determinant of developmental timing, with optimal development around 302C. RF exposure (900 MHz and 18 GHz) was described as a secondary factor that could accelerate or prolong development depending on temperature, with synergistic shortening at 250C and prolongation under suboptimal conditions.