RF Safe publishes a commentary disputing Media Bias Fact Check’s (MBFC) labeling of RF Safe as “pseudoscience” with “mixed factual reporting” and “low credibility.” The post argues MBFC mischaracterized RF Safe’s content as overstating evidence about cell phones and health, claiming RF Safe generally uses cautious, study-referencing language (e.g., “associations,” “potential risks”) and avoids asserting direct human causation. It also points to RF Safe disclaimers that the site is educational and not medical advice, and highlights its research library linking to primary studies such as NTP and Ramazzini animal findings.

This PubMed-listed paper argues that the U.S. regulatory framework for radiofrequency radiation (RFR) from wireless technologies is outdated, lacks adequate oversight and enforcement, and has not been meaningfully updated since 1996. It contends that FCC exposure limits focus on short-term, high-intensity effects and do not address long-term, low-intensity exposures, with insufficient safeguards for children, pregnancy, and other vulnerable groups. The authors also discuss alleged regulatory capture, gaps in monitoring and compliance, and propose reforms including independent research, updated safety limits, and stronger pre- and post-market surveillance.

RF Safe criticizes Media Bias Fact Check (MBFC) for labeling RF Safe as “pseudoscience” with “mixed factual reporting” and “low credibility,” arguing MBFC’s entry contains factual errors and misrepresentations. The post says RF Safe does not claim RF radiation definitively causes human disease, but instead presents precautionary interpretations of peer-reviewed studies and proposed non-thermal mechanisms. It also alleges MBFC made specific, checkable mistakes about study-linking practices and site ownership/funding, and failed to correct them after rebuttals.

RF Safe publishes a rebuttal to Media Bias Fact Check (MBFC) after MBFC labeled RF Safe as “pseudoscience” with “mixed factual reporting” and “low credibility.” The article argues MBFC made factual errors about RF Safe’s research links and ownership/funding, and says MBFC has not corrected the entry despite requests. RF Safe also defends its framing of non-thermal RF/EMF effects as precautionary and grounded in peer-reviewed literature, while criticizing what it characterizes as superficial fact-checking.

RF Safe argues that many “anti-radiation” phone cases use misleading marketing (e.g., fabric-swatch tests, vague “FCC tested” claims) and that some designs may cause phones to increase transmit power if they interfere with antennas. The article provides a checklist of red flags (magnets/metal plates, detachable shields, unclear orientation instructions) and emphasizes behavioral steps to reduce RF exposure. It promotes RF Safe’s QuantaCase as a “directional shielding” design intended to reduce exposure on the body-facing side while avoiding signal blockage that could prompt higher power output.

RF Safe argues that celebrity-branded mobile services (citing reported plans for “Beast Mobile” and the announced “Trump Mobile”) could normalize near-body, all-day phone use—especially among children—and therefore carry ethical responsibility for scaled RF exposure. The piece cites legal and scientific developments (including the 2021 Environmental Health Trust v. FCC decision, the U.S. NTP animal studies, and a WHO-commissioned systematic review) to claim the evidence base has “moved decisively” toward concern about long-term RF-EMF effects. It also promotes a proposed mechanistic framework ("S4–Mito–Spin") and suggests shifting indoor connectivity toward Li‑Fi (IEEE 802.11bb) as a harm-reduction approach.

This RF Safe commentary argues that U.S. RF exposure protections remain anchored to “thermal-only” assumptions from the 1990s despite what it describes as newer WHO-commissioned systematic reviews elevating certain animal cancer endpoints and a male fertility endpoint to “high certainty.” It contrasts these claims with a WHO-commissioned review of human observational studies that reportedly found mobile-phone RF exposure is likely not associated with increased risk of several head/brain tumors, arguing that this is often overgeneralized in public messaging. The piece calls for “prudent avoidance,” updates to FCC rules, and highlights legal and policy constraints such as federal preemption under the Telecommunications Act and a 2021 D.C. Circuit decision criticizing the FCC’s rationale for retaining its RF limits without adequate explanation.

RF Safe argues that recent WHO-linked evidence reviews have moved beyond a “thermal-only” safety narrative and that policy should respond with stronger protections. The post cites a 2025 WHO-commissioned systematic review in Environment International as concluding with “high certainty” that RF-EMF increases malignant heart schwannomas and brain gliomas in male rats, and references a 2025 corrigendum upgrading certainty for reduced pregnancy rates after male RF exposure in animal experiments. It also points to U.S. FCC rules being rooted in 1996-era assumptions and references a U.S. appellate court remand requiring the FCC to better address non-cancer harms and impacts on children and long-term exposure. The article advocates for the “Clean Ether Act” and promotes RF Safe’s proposed “S4–Mito–Spin” mechanism framework as a non-thermal explanatory model.

RF Safe argues that children’s everyday wireless exposure is primarily shaped by policy choices (laws, agency guidance, research mandates, and procurement practices) rather than by technology alone. The post promotes an “Act Now” hub that offers coordinated advocacy actions aimed at changing federal and local rules, increasing research and oversight, and shifting indoor connectivity toward alternatives such as Li‑Fi. It frames current governance as outdated and restrictive, particularly around local authority and federal agency accountability.

RF Safe promotes an “Act Now” hub intended to convert EMF safety concerns into policy and regulatory actions, emphasizing accountability and exposure reduction, especially for children. The page outlines five advocacy “levers,” including changing Section 704 of the Telecommunications Act, pressing the FCC to complete actions following a court remand, and restarting a federal electronic-product radiation program. It frames current RF oversight as outdated and insufficient for modern exposure patterns, and provides scripts to help supporters submit comments and demands into official records.

This article summarizes expert presentations and a panel discussion on radiological protection systems for ionizing and non-ionizing radiation at an international congress. It highlights that ionizing radiation protection is mature and continually revised, while non-ionizing radiation protection lacks a comparable international framework. The authors emphasize that emerging non-ionizing technologies create complex exposure scenarios and unresolved concerns about chronic and acute exposures, calling for a more cohesive and protective framework.

This animal study tested whether short-term 28 GHz (5G-band) millimeter-wave exposure modifies doxorubicin-induced cardiotoxicity in male rats and whether vitamin C mitigates effects. Co-exposure to 28 GHz EMR was reported to worsen several indices of DOX-related cardiac injury (including CAT reduction, increased BAX expression, and QT prolongation), while vitamin C provided partial attenuation. The authors emphasize that results are limited to a short-duration preclinical model and that human relevance remains preliminary.

This magazine article discusses the rising incidence of thyroid cancer and raises the possibility of an association with increased smartphone use and related RF EMF exposure near the head and neck. It characterizes EMF exposure from personal electronics as a growing public health concern. The piece calls for more research, monitoring, and public awareness, and mentions precautionary measures.

This exposure assessment measured outdoor micro-environment RF-EMF levels in daily-life settings across urban and suburban locations in Japan using a portable device (50 MHz–6 GHz) with GPS. Reported exposure levels were higher in urban areas, with railway stations showing the highest levels among the environments measured. The authors emphasize the need for further comprehensive studies and frame prolonged RF-EMF exposure as an ongoing public health concern.

This animal experiment assessed GSM-modulated 3.5 GHz RF exposure in male Wistar rats and reported hormonal, oxidative, and histological changes consistent with testicular impairment. RF exposure was associated with lower testosterone, LH, and FSH, higher oxidative stress (increased MDA and TOS), and degenerative testicular histology. Coenzyme Q10 supplementation partially mitigated several reported changes. The authors caution against generalizing these results to FR1 5G NR signals and call for further research.

This in vitro study examined strictly non-thermal, GSM-like 3.5 GHz RF-EMF exposure in cultured mouse dorsal root ganglion neurons for 1–24 hours. The authors report time-dependent reductions in cell viability alongside increased ROS and changes consistent with mitochondria-mediated apoptosis (e.g., Bax/caspase-3 up, cytochrome c release, Bcl-2 down) and increased p75NTR. They conclude these findings provide mechanistic evidence of peripheral neuronal vulnerability to mid-band RF exposure and call for further in vivo research.

This rat study examined 2.45 GHz WLAN-like EMF exposure (3 V/m; SAR 0.00208 W/kg) for 1 hour/day over 60 days and assessed testicular morphology and VEGF-related markers. The abstract reports increased VEGFA gene expression and protein levels in exposed animals, with no change in HIF1A expression. It also reports multiple histological changes interpreted as testicular damage in the exposed group.

This in vitro study tested whether 1800 MHz RF-EMF modifies chemically induced DNA damage in mouse embryonic fibroblasts under non-thermal exposure conditions. RF-EMF alone did not produce detectable DNA damage and did not significantly enhance damage from hydrogen peroxide, 4NQO, or cadmium. In contrast, co-exposure with hexavalent chromium (Cr(VI)) was reported to synergistically increase DNA damage, suggesting a selective co-genotoxic interaction under specific chemical conditions.