An RF Safe article argues that “non-thermal” RF/ELF electromagnetic fields from phones, Wi‑Fi, and 5G cause widespread biological harm and that regulators and industry have misled the public by focusing on heating-based safety limits. It claims 2025 is a “tipping point,” citing WHO-commissioned reviews, animal studies (e.g., NTP/Ramazzini), a “Frontiers review,” and ICBE-EMF statements as evidence of cancer and other health risks. The piece frames the issue as urgent and settled, calling for public action and policy change, but presents these conclusions in advocacy language without providing verifiable study details in the excerpt.

An RF Safe article argues that, as of 2025, evidence is “decisive” that man-made radiofrequency (RF) fields can cause cancer and other biological harm, and that non-thermal mechanisms are now established. It cites animal studies (including NTP and Ramazzini), a 2025 WHO-commissioned systematic review (as described by the author), and proposed mechanisms involving voltage-gated ion channels, oxidative stress, and radical-pair/spin chemistry. The piece calls for updated safety standards that consider modulation and tissue vulnerability, while stating it is “not a call for panic.”

RF Safe presents the “Herzification / Bioelectric Fidelity Hypothesis,” arguing that modern RF/EMF exposure has rapidly altered the human electromagnetic environment and may degrade biological electrical signaling (“bioelectric fidelity”). The post frames this as an “evidence-anchored hypothesis” that could help explain a wide range of outcomes (e.g., cancer, infertility, ADHD-like traits, some autism phenotypes, emotional dysregulation), while acknowledging it is not definitive proof. It also cites Heinrich Hertz’s illness as a suggestive historical anecdote and references proposed non-thermal interaction mechanisms involving voltage-gated ion channels.

This RF Safe article argues that a common biological mechanism links RF/ELF exposure to downstream outcomes such as cancer, infertility, and autoimmune dysfunction. It proposes a causal chain in which RF/ELF fields disrupt S4 voltage-sensor timing in voltage-gated ion channels, altering calcium signaling and triggering mitochondrial reactive oxygen species (ROS) that lead to tissue-specific damage. The piece cites mechanistic researchers and references major animal studies and WHO-commissioned systematic reviews, but presents the argument as a unifying narrative rather than a new peer-reviewed study.

RF Safe publishes an advocacy guide arguing that current wireless RF/MW exposure limits are “thermal-only,” outdated since 1996, and insufficient to address claimed non-thermal biological effects from pulsed/modulated signals. The guide summarizes mechanistic arguments (e.g., voltage-gated ion channel timing disruption), cites animal studies and reviews it says link RF exposure to cancer and other harms, and calls for regulatory and technological reforms (including Li‑Fi) plus exposure-reduction strategies. The piece frames the issue as urgent and precautionary, presenting its synthesis as evidence-grounded but primarily as advocacy rather than a single new study.

No abstract was provided. From the title and supplied overview, this paper critiques the Mevissen et al. systematic review on RF-EMF exposure and cancer in animal studies, asserting that methodological and analytical flaws led to misjudgment of the evidence. The provided text frames the topic as requiring careful analysis to avoid underestimating potential health risks.

This systematic review evaluated RF EMF exposure and cancer outcomes in experimental animals, including chronic cancer bioassays and tumor-promotion designs. Across 52 included studies, the authors report high certainty of evidence for increased malignant heart schwannomas and gliomas in male rats, and moderate certainty for increased risks of several other tumor types. Many other organ systems showed no or minimal evidence of carcinogenic effects, and the authors note challenges in translating animal findings to human risk assessment due to exposure and mechanistic uncertainties.