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    <title>Reviewed Papers — Evidence: Low</title>
    <link>https://rfsafe.org/mel/papers_class.php?evidence=low</link>
    <description>Reviewed papers with evidence strength: Low.</description>
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    <item>
      <title>Exposure to hexavalent chromium and 1800 MHz electromagnetic radiation can synergistically induce intracellular DNA damage in mouse embryonic fibroblasts</title>
      <link>https://rfsafe.org/mel/paper.php?id=2455</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=2455</guid>
      <pubDate>Thu, 12 Mar 2026 00:00:00 +0000</pubDate>
      <category>effect:mixed</category>
      <category>evidence:low</category>
      <category>year:2026</category>
      <category>study_type:in_vitro</category>
      <description><![CDATA[<p><strong>Effect:</strong> Mixed &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> in_vitro</p>
<p><small>Biochem Biophys Res Commun · 2026 · Manual</small></p>
<p>Under standardized non-thermal conditions, 1800 MHz RF-EMF exposure alone did not induce detectable DNA damage in mouse embryonic fibroblasts. RF-EMF did not significantly enhance DNA damage caused by H2O2, 4NQO, or Cd2+, but co-exposure with hexavalent chromium [Cr(VI)] produced a significant synergistic increase in DNA damage.</p>
<p><small>DOI: 10.1016/j.bbrc.2026.153360 · PMID: 41619510</small></p>
<p><small><strong>Study URL:</strong> <a href="https://pubmed.ncbi.nlm.nih.gov/41619510/" target="_blank" rel="noopener">https://pubmed.ncbi.nlm.nih.gov/41619510/</a></small></p>]]></description>
    </item>
    <item>
      <title>Human auditory system response to modulated electromagnetic energy</title>
      <link>https://rfsafe.org/mel/paper.php?id=6796</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=6796</guid>
      <pubDate>Mon, 01 Jan 1962 00:00:00 +0000</pubDate>
      <category>effect:mixed</category>
      <category>evidence:low</category>
      <category>year:1962</category>
      <category>study_type:other</category>
      <description><![CDATA[<p><strong>Effect:</strong> Mixed &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> other</p>
<p><small>J. Appl. Physiol. · 1962 · Manual</small></p>
<p>The paper reports that modulated RF electromagnetic energy induced perception of sounds in normal and deaf humans at distances of several hundred feet from the antenna when the transmitter was turned on. The effect depended on carrier frequency, modulation, and peak power density; under approximately 80 dB acoustic noise, a peak power density of about 275 mW/cm2 was needed at carrier frequencies of 425 and 1,310 MHz, while average power density could be as low as 400 pW/cm2.</p>
<p><small><strong>Study URL:</strong> <a href="https://mriquestions.com/uploads/3/4/5/7/34572113/auditory_frey_rf_hearing_jappl.1962.17.4.689.pdf" target="_blank" rel="noopener">https://mriquestions.com/uploads/3/4/5/7/34572113/auditory_frey_rf_hearing_jappl.1962.17.4.689.pdf</a></small></p>]]></description>
    </item>
    <item>
      <title>Effect of ultra‑wide‑band electromagnetic pulses on blood‑brain barrier permeability in rats</title>
      <link>https://rfsafe.org/mel/paper.php?id=6795</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=6795</guid>
      <pubDate>Wed, 01 Jan 2020 00:00:00 +0000</pubDate>
      <category>effect:harm</category>
      <category>evidence:low</category>
      <category>year:2020</category>
      <category>study_type:animal</category>
      <description><![CDATA[<p><strong>Effect:</strong> Harm &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> animal</p>
<p><small>Mol Med Rep · 2020 · Manual</small></p>
<p>In rats, BBB permeability increased immediately after ultra-wide-band electromagnetic pulse exposure, peaked at 3-6 h, and returned to baseline by 24 h. BBB opening was observed at 200 and 400 kV/m but not at 50 kV/m, and ZO-1 expression decreased at 3 and 6 h without an observed change in ZO-1 distribution.</p>
<p><small>DOI: 10.3892/mmr.2020.11382 · PMID: 32945403</small></p>
<p><small><strong>Study URL:</strong> <a href="https://pubmed.ncbi.nlm.nih.gov/32945403/" target="_blank" rel="noopener">https://pubmed.ncbi.nlm.nih.gov/32945403/</a></small></p>]]></description>
    </item>
    <item>
      <title>[Electromagnetic bioengineering]</title>
      <link>https://rfsafe.org/mel/paper.php?id=6793</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=6793</guid>
      <pubDate>Thu, 01 Jan 1998 00:00:00 +0000</pubDate>
      <category>effect:mixed</category>
      <category>evidence:low</category>
      <category>year:1998</category>
      <category>study_type:review</category>
      <description><![CDATA[<p><strong>Effect:</strong> Mixed &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> review</p>
<p><small>Biofizika · 1998 · Manual</small></p>
<p>This paper generalises experimental and theoretical studies on the combined action of weak electrical fields and constant and variable low-frequency magnetic fields on physicochemical and biological systems. It reports that weak magnetic fields with parameters close to the geomagnetic field selectively affect aqueous solutions of organic molecules by initiating chemical reactivity and ion conductivity processes.</p>
<p><small>PMID: 9783064</small></p>
<p><small><strong>Study URL:</strong> <a href="https://pubmed.ncbi.nlm.nih.gov/9783064/" target="_blank" rel="noopener">https://pubmed.ncbi.nlm.nih.gov/9783064/</a></small></p>]]></description>
    </item>
    <item>
      <title>Electromagnetic Fields, Pulsed Radiofrequency Radiation, and Epigenetics: How Wireless Technologies May Affect Childhood Development</title>
      <link>https://rfsafe.org/mel/paper.php?id=6792</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=6792</guid>
      <pubDate>Mon, 01 Jan 2018 00:00:00 +0000</pubDate>
      <category>effect:harm</category>
      <category>evidence:low</category>
      <category>year:2018</category>
      <category>study_type:review</category>
      <description><![CDATA[<p><strong>Effect:</strong> Harm &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> review</p>
<p><small>Child Dev · 2018 · Manual</small></p>
<p>This review profiles epigenetic studies related to exposure to mobile phones and other wireless devices producing EMF and pulsed radiofrequency radiation. The abstract states that numerous studies have reported harmful health impacts and neurodevelopmental/neurobehavioral symptoms in young people, with epigenetic mechanisms and DNA damage described as likely contributors.</p>
<p><small>DOI: 10.1111/cdev.12824 · PMID: 28504324</small></p>
<p><small><strong>Study URL:</strong> <a href="https://pubmed.ncbi.nlm.nih.gov/28504324/" target="_blank" rel="noopener">https://pubmed.ncbi.nlm.nih.gov/28504324/</a></small></p>]]></description>
    </item>
    <item>
      <title>Autism-relevant social abnormalities in mice exposed perinatally to extremely low frequency electromagnetic fields</title>
      <link>https://rfsafe.org/mel/paper.php?id=4683</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=4683</guid>
      <pubDate>Wed, 01 Jan 2014 00:00:00 +0000</pubDate>
      <category>effect:mixed</category>
      <category>evidence:low</category>
      <category>year:2014</category>
      <category>study_type:animal</category>
      <description><![CDATA[<p><strong>Effect:</strong> Mixed &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> animal</p>
<p><small>Int J Dev Neurosci · 2014 · Manual</small></p>
<p>Perinatal ELF-EMF-exposed male BALB/C mice showed impaired sociability and reduced preference for social novelty, along with decreased exploratory activity. Anxiety-like behavior, locomotion, motor coordination, and olfaction were reported as normal in exposed mice.</p>
<p><small>DOI: 10.1016/j.ijdevneu.2014.06.010 · PMID: 24970316</small></p>
<p><small><strong>Study URL:</strong> <a href="https://pubmed.ncbi.nlm.nih.gov/24970316/" target="_blank" rel="noopener">https://pubmed.ncbi.nlm.nih.gov/24970316/</a></small></p>]]></description>
    </item>
    <item>
      <title>Use of mobile phones and cordless phones is associated with increased risk for glioma and acoustic neuroma</title>
      <link>https://rfsafe.org/mel/paper.php?id=6788</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=6788</guid>
      <pubDate>Tue, 01 Jan 2013 00:00:00 +0000</pubDate>
      <category>effect:harm</category>
      <category>evidence:low</category>
      <category>year:2013</category>
      <category>study_type:review</category>
      <description><![CDATA[<p><strong>Effect:</strong> Harm &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> review</p>
<p><small>Pathophysiology · 2013 · Manual</small></p>
<p>This paper provides an overview of epidemiological evidence and reports a meta-analysis of Hardell group and Interphone results for mobile phone use. In the meta-analysis, higher odds were reported for glioma in the temporal lobe with latency ≥10 years (OR=1.71, 95% CI=1.04-2.81) and for ipsilateral cumulative mobile phone use ≥1640 h (OR=2.29, 95% CI=1.56-3.37); acoustic neuroma risk was also elevated for ipsilateral cumulative use ≥1640 h (OR=2.55, 95% CI=1.50-4.40). The abstract also states that cordless phone use increased risk for glioma and acoustic neuroma in Hardell group studies.</p>
<p><small>DOI: 10.1016/j.pathophys.2012.11.001 · PMID: 23261330</small></p>
<p><small><strong>Study URL:</strong> <a href="https://pubmed.ncbi.nlm.nih.gov/23261330/" target="_blank" rel="noopener">https://pubmed.ncbi.nlm.nih.gov/23261330/</a></small></p>]]></description>
    </item>
    <item>
      <title>Mobile phones, cordless phones and the risk for brain tumours</title>
      <link>https://rfsafe.org/mel/paper.php?id=6787</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=6787</guid>
      <pubDate>Thu, 01 Jan 2009 00:00:00 +0000</pubDate>
      <category>effect:harm</category>
      <category>evidence:low</category>
      <category>year:2009</category>
      <category>study_type:case_control</category>
      <description><![CDATA[<p><strong>Effect:</strong> Harm &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> case_control</p>
<p><small>Int J Oncol · 2009 · Manual</small></p>
<p>In two Swedish case-control studies, ipsilateral use of mobile and cordless phones was associated with higher odds of astrocytoma, especially with &gt;10 years latency (mobile phone OR 3.3, 95% CI 2.0-5.4; cordless phone OR 5.0, 95% CI 2.3-11). For acoustic neuroma, the highest odds were reported for ipsilateral mobile phone use with &gt;10 years latency (OR 3.0, 95% CI 1.4-6.2), while the cordless phone estimate was imprecise (OR 2.3, 95% CI 0.6-8.8). Highest risks were also reported among subjects with first use before age 20 years.</p>
<p><small>DOI: 10.3892/ijo_00000307 · PMID: 19513546</small></p>
<p><small><strong>Study URL:</strong> <a href="https://pubmed.ncbi.nlm.nih.gov/19513546/" target="_blank" rel="noopener">https://pubmed.ncbi.nlm.nih.gov/19513546/</a></small></p>]]></description>
    </item>
    <item>
      <title>Pooled analysis of case-control studies on malignant brain tumours and the use of mobile and cordless phones including living and deceased subjects</title>
      <link>https://rfsafe.org/mel/paper.php?id=6786</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=6786</guid>
      <pubDate>Sat, 01 Jan 2011 00:00:00 +0000</pubDate>
      <category>effect:harm</category>
      <category>evidence:low</category>
      <category>year:2011</category>
      <category>study_type:policy</category>
      <description><![CDATA[<p><strong>Effect:</strong> Harm &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> policy</p>
<p><small>Int J Oncol · 2011 · Manual</small></p>
<p>This pooled analysis of case-control studies reported increased odds of glioma associated with both mobile and cordless phone use. Risk increased with longer latency and greater cumulative hours of use, with the highest reported risk for astrocytoma in the &gt;10 year latency group and among those with first wireless phone use before age 20.</p>
<p><small>DOI: 10.3892/ijo.2011.947 · PMID: 21331446</small></p>
<p><small><strong>Study URL:</strong> <a href="https://pubmed.ncbi.nlm.nih.gov/21331446/" target="_blank" rel="noopener">https://pubmed.ncbi.nlm.nih.gov/21331446/</a></small></p>]]></description>
    </item>
    <item>
      <title>Mobile phone and cordless phone use and the risk for glioma - Analysis of pooled case-control studies in Sweden, 1997-2003 and 2007-2009</title>
      <link>https://rfsafe.org/mel/paper.php?id=6785</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=6785</guid>
      <pubDate>Thu, 01 Jan 2015 00:00:00 +0000</pubDate>
      <category>effect:harm</category>
      <category>evidence:low</category>
      <category>year:2015</category>
      <category>study_type:policy</category>
      <description><![CDATA[<p><strong>Effect:</strong> Harm &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> policy</p>
<p><small>Pathophysiology · 2015 · Manual</small></p>
<p>In this pooled analysis of two Swedish case-control studies, mobile phone use was associated with increased glioma risk overall (OR 1.3, 95% CI 1.1-1.6), rising to OR 3.0 (95% CI 1.7-5.2) in the &gt;25-year latency group. Cordless phone use was also associated with increased glioma risk (OR 1.4, 95% CI 1.1-1.7), with higher odds for longer latency and ipsilateral use; the highest risk was reported for temporal lobe glioma.</p>
<p><small>DOI: 10.1016/j.pathophys.2014.10.001 · PMID: 25466607</small></p>
<p><small><strong>Study URL:</strong> <a href="https://pubmed.ncbi.nlm.nih.gov/25466607/" target="_blank" rel="noopener">https://pubmed.ncbi.nlm.nih.gov/25466607/</a></small></p>]]></description>
    </item>
    <item>
      <title>Is Cellphone Carrying Below the Waist (Exposure to Non-Ionizing Radiation) Contributing to the Rapid Rise in Early-Onset Colorectal Cancer?</title>
      <link>https://rfsafe.org/mel/paper.php?id=2426</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=2426</guid>
      <pubDate>Mon, 01 Jan 2024 00:00:00 +0000</pubDate>
      <category>effect:harm</category>
      <category>evidence:low</category>
      <category>year:2024</category>
      <category>study_type:case_control</category>
      <description><![CDATA[<p><strong>Effect:</strong> Harm &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> case_control</p>
<p><small>ISEE Conference Abstracts · 2024 · Manual</small></p>
<p>Individuals carrying a phone below the waist were four times more likely to develop early-onset colorectal cancer. Ipsilateral carrying on the left side for more than 30,000 hours was associated with a 12-fold increased risk of tumor on that side. Contralateral carrying showed only a slight increase in risk.</p>
<p><small>DOI: 10.1289/isee.2024.1903</small></p>
<p><small><strong>Study URL:</strong> <a href="https://www.researchgate.net/publication/383147719_Is_Cellphone_Carrying_Below_the_Waist_Exposure_to_Non-Ionizing_Radiation_Contributing_to_the_Rapid_Rise_in_Early-Onset_Colorectal_Cancer" target="_blank" rel="noopener">https://www.researchgate.net/publication/383147719_Is_Cellphone_Carrying_Below_the_Waist_Exposure_to_Non-Ionizing_Radiation_Contributing_to_the_Rapid_Rise_in_Early-Onset_Colorectal_Cancer</a></small></p>]]></description>
    </item>
    <item>
      <title>PROBABLE EFFECT OF COMMUNICATION TOWERS ELECTROMAGNETIC WAVES ON NEARBY AREAS RESIDENTS DNA INTEGRITY</title>
      <link>https://rfsafe.org/mel/paper.php?id=6780</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=6780</guid>
      <pubDate>Wed, 01 Jan 2025 00:00:00 +0000</pubDate>
      <category>effect:harm</category>
      <category>evidence:low</category>
      <category>year:2025</category>
      <category>study_type:cross_sectional</category>
      <description><![CDATA[<p><strong>Effect:</strong> Harm &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> cross_sectional</p>
<p><small>Iraqi Journal of Market Research and Consumer Protection · 2025 · Manual</small></p>
<p>Serum 8-OHdG was higher in participants living near communication towers (3.9) compared with the control group living far from towers (0.46).</p>
<p><small>DOI: 10.28936/jmracpc17.1.2025.(13</small></p>
<p><small><strong>Study URL:</strong> <a href="https://doi.org/10.28936/jmracpc17.1.2025.(13)" target="_blank" rel="noopener">https://doi.org/10.28936/jmracpc17.1.2025.(13)</a></small></p>]]></description>
    </item>
    <item>
      <title>Effect of cell phone exposure on physiologic and hematologic parameters of male medical students of Bijapur (Karnataka) with reference to serum lipid profile</title>
      <link>https://rfsafe.org/mel/paper.php?id=6779</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=6779</guid>
      <pubDate>Fri, 01 Jan 2010 00:00:00 +0000</pubDate>
      <category>effect:mixed</category>
      <category>evidence:low</category>
      <category>year:2010</category>
      <category>study_type:cross_sectional</category>
      <description><![CDATA[<p><strong>Effect:</strong> Mixed &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> cross_sectional</p>
<p><small>J Basic Clin Physiol Pharmacol · 2010 · Manual</small></p>
<p>No significant differences between long-term exposed and never-exposed groups were reported for basal heart rate, systolic blood pressure, SpO2(%), or various hematologic parameters. During an acute 1-minute 900 MHz call, peak heart rate increased significantly in the long-term exposed group compared with the never-exposed group, and peak SpO2(%) decreased significantly in the never-exposed group compared with the long-term exposed group. Serum total cholesterol, VLDL-cholesterol, and triglycerides were significantly higher in the long-term exposed group, described as a mild alteration of lipid profile.</p>
<p><small>DOI: 10.1515/jbcpp.2010.21.2.201 · PMID: 20853601</small></p>
<p><small><strong>Study URL:</strong> <a href="https://pubmed.ncbi.nlm.nih.gov/20853601/" target="_blank" rel="noopener">https://pubmed.ncbi.nlm.nih.gov/20853601/</a></small></p>]]></description>
    </item>
    <item>
      <title>Effects of electromagnetic fields on serum lipids in workers of a power plant</title>
      <link>https://rfsafe.org/mel/paper.php?id=4741</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=4741</guid>
      <pubDate>Fri, 01 Jan 2016 00:00:00 +0000</pubDate>
      <category>effect:harm</category>
      <category>evidence:low</category>
      <category>year:2016</category>
      <category>study_type:cross_sectional</category>
      <description><![CDATA[<p><strong>Effect:</strong> Harm &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> cross_sectional</p>
<p><small>Environ Sci Pollut Res Int · 2016 · Manual</small></p>
<p>In this cross-sectional study of power plant workers, occupational EMF exposure (including RF and ELF) was associated with higher LDL-c (β = 0.17 mmol/L, P = 0.022). In the high-exposure group, longer employment duration, longer daily exposure duration, and higher mobile phone or electric fee per month were associated with higher total cholesterol, LDL-c, or triglycerides; decreased HDL-c was reported only in the high-exposure group with higher mobile phone fee per month. Similar patterns were reported in 544 participants with serum lipid data available in 2010.</p>
<p><small>DOI: 10.1007/s11356-015-5500-9 · PMID: 26423285</small></p>
<p><small><strong>Study URL:</strong> <a href="https://pubmed.ncbi.nlm.nih.gov/26423285/" target="_blank" rel="noopener">https://pubmed.ncbi.nlm.nih.gov/26423285/</a></small></p>]]></description>
    </item>
    <item>
      <title>Exposure of Man to Magnetic Fields Alternating at Extremely Low Frequency</title>
      <link>https://rfsafe.org/mel/paper.php?id=6778</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=6778</guid>
      <pubDate>Mon, 01 Jan 1973 00:00:00 +0000</pubDate>
      <category>effect:unclear</category>
      <category>evidence:low</category>
      <category>year:1973</category>
      <category>study_type:randomized_trial</category>
      <description><![CDATA[<p><strong>Effect:</strong> Unclear &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> randomized_trial</p>
<p><small>1973 · Manual</small></p>
<p>No definite effects were linked to ELF magnetic field exposure except an increase in serum triglycerides 24 to 40 hours after exposure in 9 of 10 exposed subjects, not seen in controls. Cholesterol and chylomicrons remained stable. Small sample size and confounding factors limit conclusions.</p>
<p><small><strong>Study URL:</strong> <a href="https://apps.dtic.mil/sti/tr/pdf/AD0770140.pdf" target="_blank" rel="noopener">https://apps.dtic.mil/sti/tr/pdf/AD0770140.pdf</a></small></p>]]></description>
    </item>
    <item>
      <title>Non-chemical signalling between mitochondria.</title>
      <link>https://rfsafe.org/mel/paper.php?id=6775</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=6775</guid>
      <pubDate>Sun, 01 Jan 2023 00:00:00 +0000</pubDate>
      <category>effect:mixed</category>
      <category>evidence:low</category>
      <category>year:2023</category>
      <category>study_type:in_vitro</category>
      <description><![CDATA[<p><strong>Effect:</strong> Mixed &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> in_vitro</p>
<p><small>Frontiers in physiology · 2023 · PubMed</small></p>
<p>Mitochondria stressed with the electron transport chain inhibitor antimycin in one cuvette altered respiration of mitochondria in an adjacent, chemically and physically separate cuvette, decreasing oxygen consumption compared to control (p &lt; 0.0001 for both MCF7 and MCF10A mitochondria). The effect depended on mitochondrial origin (cancer vs non-cancer) and the presence of ambient light, and the design suggests a light-based mechanism.</p>
<p><small>DOI: 10.3389/fphys.2023.1268075 · PMID: 37811497</small></p>
<p><small><strong>Study URL:</strong> <a href="https://pubmed.ncbi.nlm.nih.gov/37811497/" target="_blank" rel="noopener">https://pubmed.ncbi.nlm.nih.gov/37811497/</a></small></p>]]></description>
    </item>
    <item>
      <title>Exploring Intercellular Dynamics: Ultra-Weak Biophoton Emission as a Novel Indicator of Altered Cell Functions and Disease in Oligospermia Mice.</title>
      <link>https://rfsafe.org/mel/paper.php?id=6773</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=6773</guid>
      <pubDate>Sun, 01 Jan 2023 00:00:00 +0000</pubDate>
      <category>effect:unclear</category>
      <category>evidence:low</category>
      <category>year:2023</category>
      <category>study_type:animal</category>
      <description><![CDATA[<p><strong>Effect:</strong> Unclear &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> animal</p>
<p><small>Journal of lasers in medical sciences · 2023 · PubMed</small></p>
<p>Twenty male mice were assigned to control and busulfan groups and assessed 5 weeks after busulfan injection. The abstract reports significant differences between groups in ultra-weak biophoton emission, with statements that it was higher in the busulfan group and also that it was reduced in oligospermia mice; the direction is therefore inconsistent within the abstract. The busulfan (oligospermia) group showed decreased testis volume, seminiferous tubule length, and number of testicular cells, and increased apoptotic cells by TUNEL assay.</p>
<p><small>DOI: 10.34172/jlms.2023.65 · PMID: 38318218</small></p>
<p><small><strong>Study URL:</strong> <a href="https://pubmed.ncbi.nlm.nih.gov/38318218/" target="_blank" rel="noopener">https://pubmed.ncbi.nlm.nih.gov/38318218/</a></small></p>]]></description>
    </item>
    <item>
      <title>Red and near-infrared light treatment can change the intensity of biophoton emissions in cell culture.</title>
      <link>https://rfsafe.org/mel/paper.php?id=6771</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=6771</guid>
      <pubDate>Wed, 01 Jan 2025 00:00:00 +0000</pubDate>
      <category>effect:mixed</category>
      <category>evidence:low</category>
      <category>year:2025</category>
      <category>study_type:in_vitro</category>
      <description><![CDATA[<p><strong>Effect:</strong> Mixed &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> in_vitro</p>
<p><small>Scientific reports · 2025 · PubMed</small></p>
<p>In Neuro-2a cells and astrocytes, baseline biophoton emissions were low (~12 photons/s) and similar between cell types. Stress altered emissions depending on the toxin (sodium troclosene increased emissions; rotenone had a more limited impact). Red/near-infrared light did not influence emissions in healthy cells but did influence emissions under stress, particularly with sodium troclosene; emission patterns did not relate uniformly to ATP and ROS changes under the experimental timelines.</p>
<p><small>DOI: 10.1038/s41598-025-22344-0 · PMID: 41188307</small></p>
<p><small><strong>Study URL:</strong> <a href="https://pubmed.ncbi.nlm.nih.gov/41188307/" target="_blank" rel="noopener">https://pubmed.ncbi.nlm.nih.gov/41188307/</a></small></p>]]></description>
    </item>
    <item>
      <title>Non-invasive modulation of brain activity and behavior by transcranial radio frequency stimulation.</title>
      <link>https://rfsafe.org/mel/paper.php?id=6768</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=6768</guid>
      <pubDate>Thu, 01 Jan 2026 00:00:00 +0000</pubDate>
      <category>effect:mixed</category>
      <category>evidence:low</category>
      <category>year:2026</category>
      <category>study_type:animal</category>
      <description><![CDATA[<p><strong>Effect:</strong> Mixed &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> animal</p>
<p><small>Brain stimulation · 2026 · PubMed</small></p>
<p>A custom 945 MHz TRFS system produced localized brain heating in mice. In pristine mode, RF-induced temperature rises caused dose-dependent suppression of cortical PV interneuron activity and an ipsilateral rotational bias. In RF-genetics mode (TRPV1 overexpression), RF stimulation produced temperature-dependent excitation once local temperature change exceeded approximately 1.5°C and reversed rotational bias to contralateral.</p>
<p><small>DOI: 10.1016/j.brs.2026.103032 · PMID: 41548801</small></p>
<p><small><strong>Study URL:</strong> <a href="https://pubmed.ncbi.nlm.nih.gov/41548801/" target="_blank" rel="noopener">https://pubmed.ncbi.nlm.nih.gov/41548801/</a></small></p>]]></description>
    </item>
    <item>
      <title>Impact of mobile phone use on the brain activity: Audio call vs video call.</title>
      <link>https://rfsafe.org/mel/paper.php?id=6767</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=6767</guid>
      <pubDate>Thu, 01 Jan 2026 00:00:00 +0000</pubDate>
      <category>effect:harm</category>
      <category>evidence:low</category>
      <category>year:2026</category>
      <category>study_type:other</category>
      <description><![CDATA[<p><strong>Effect:</strong> Harm &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> other</p>
<p><small>Acta psychologica · 2026 · PubMed</small></p>
<p>EEG-based indices indicated that video calls produced significantly higher cognitive load than audio calls (example TAR means reported for 3-minute sessions; p&lt;0.05) and higher mental fatigue (example values reported for 4-minute sessions). Effects were stronger with longer call duration, and ML classification distinguished call conditions with ROC AUC &gt; 0.90.</p>
<p><small>DOI: 10.1016/j.actpsy.2025.106160 · PMID: 41601117</small></p>
<p><small><strong>Study URL:</strong> <a href="https://pubmed.ncbi.nlm.nih.gov/41601117/" target="_blank" rel="noopener">https://pubmed.ncbi.nlm.nih.gov/41601117/</a></small></p>]]></description>
    </item>
    <item>
      <title>Nonthermal effects of radiofrequency-field exposure on calcium dynamics in stem cell-derived neuronal cells: elucidation of calcium pathways</title>
      <link>https://rfsafe.org/mel/paper.php?id=6762</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=6762</guid>
      <pubDate>Tue, 01 Jan 2008 00:00:00 +0000</pubDate>
      <category>effect:harm</category>
      <category>evidence:low</category>
      <category>year:2008</category>
      <category>study_type:in_vitro</category>
      <description><![CDATA[<p><strong>Effect:</strong> Harm &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> in_vitro</p>
<p><small>Radiat Res · 2008 · Manual</small></p>
<p>Real-time measurements showed increased cytosolic Ca(2+) and increased Ca(2+) spiking in response to nonthermal RF exposure from 700–1100 MHz. Example given: 800 MHz at 0.5 W/kg increased spikes to 15.7±0.8 vs ~5 spikes/cell/60 min in controls (P&lt;0.05). The increase depended on frequency but not on SAR between 0.5–5 W/kg; pharmacological agents implicated N-type Ca(2+) channels and phospholipase C, and microfilament disruption prevented Ca(2+) spikes.</p>
<p><small>DOI: 10.1667/rr1118.1 · PMID: 18302487</small></p>
<p><small><strong>Study URL:</strong> <a href="https://pubmed.ncbi.nlm.nih.gov/18302487/" target="_blank" rel="noopener">https://pubmed.ncbi.nlm.nih.gov/18302487/</a></small></p>]]></description>
    </item>
    <item>
      <title>Predictors of Risk Perception Among General Practitioners and Paediatricians Concerning Potential Health Effects of Exposure to Electromagnetic Fields</title>
      <link>https://rfsafe.org/mel/paper.php?id=6761</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=6761</guid>
      <pubDate>Thu, 01 Jan 2026 00:00:00 +0000</pubDate>
      <category>effect:unclear</category>
      <category>evidence:low</category>
      <category>year:2026</category>
      <category>study_type:cross_sectional</category>
      <description><![CDATA[<p><strong>Effect:</strong> Unclear &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> cross_sectional</p>
<p><small>Bioelectromagnetics · 2026 · Manual</small></p>
<p>In a 2023 cross-sectional survey of 292 German GPs and paediatricians, 31% reported believing in health issues as a consequence of EMF exposure. Higher conspiracy belief was associated with higher EMF risk perception (OR 2.92; 95% CI 1.81–4.13), while higher trust in WHO (OR 0.57; 95% CI 0.35–0.82) and the Federal Office for Radiation Protection (OR 0.50; 95% CI 0.28–0.76) was associated with lower EMF risk perception.</p>
<p><small>DOI: 10.1002/bem.70047 · PMID: 41744411</small></p>
<p><small><strong>Study URL:</strong> <a href="https://pubmed.ncbi.nlm.nih.gov/41744411/" target="_blank" rel="noopener">https://pubmed.ncbi.nlm.nih.gov/41744411/</a></small></p>]]></description>
    </item>
    <item>
      <title>Extremely low frequency electromagnetic fields and cancer: How source of funding affects results</title>
      <link>https://rfsafe.org/mel/paper.php?id=1725</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=1725</guid>
      <pubDate>Tue, 01 Jan 2019 00:00:00 +0000</pubDate>
      <category>effect:harm</category>
      <category>evidence:low</category>
      <category>year:2019</category>
      <category>study_type:review</category>
      <description><![CDATA[<p><strong>Effect:</strong> Harm &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> review</p>
<p><small>Environ Res · 2019 · Manual</small></p>
<p>This review reports that several meta-analyses since about 2000 found significant associations between 50-60 Hz magnetic field exposure and childhood leukemia. It states that government or independent studies more often reported significant or elevated risks, whereas industry-supported studies generally did not, and concludes that there is strong evidence for increased risks of adult leukemia, breast cancer, and brain cancer with excessive magnetic field exposure.</p>
<p><small>DOI: 10.1016/j.envres.2019.108688</small></p>
<p><small><strong>Study URL:</strong> <a href="https://www.ncbi.nlm.nih.gov/pubmed/31476684" target="_blank" rel="noopener">https://www.ncbi.nlm.nih.gov/pubmed/31476684</a></small></p>]]></description>
    </item>
    <item>
      <title>Mobile telephony radiation exerts genotoxic action and significantly enhances the effects of gamma radiation in human cells</title>
      <link>https://rfsafe.org/mel/paper.php?id=179</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=179</guid>
      <pubDate>Mon, 01 Jan 2024 00:00:00 +0000</pubDate>
      <category>effect:harm</category>
      <category>evidence:low</category>
      <category>year:2024</category>
      <category>study_type:other</category>
      <description><![CDATA[<p><strong>Effect:</strong> Harm &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> other</p>
<p><small>Gen Physiol Biophys · 2024 · Manual</small></p>
<p>Human peripheral blood lymphocytes exposed to mobile telephony electromagnetic fields showed chromosomal damage. Pre-exposure to a single 15-minute mobile telephony EMF exposure before gamma radiation (0.1, 0.3, or 0.5 Gy) increased chromosomal aberrations significantly more than the sum of aberrations from the individual stressors.</p>
<p><small>DOI: 10.4149/gpb_2023036</small></p>
<p><small><strong>Study URL:</strong> <a href="http://www.elis.sk/download_file.php?product_id=8263&amp;amp;session_id=ef2ijbhbn0hevd1s33q2nvkrp6" target="_blank" rel="noopener">http://www.elis.sk/download_file.php?product_id=8263&amp;amp;session_id=ef2ijbhbn0hevd1s33q2nvkrp6</a></small></p>]]></description>
    </item>
    <item>
      <title>Altered development in rodent brain cells after 900 MHz radiofrequency exposure</title>
      <link>https://rfsafe.org/mel/paper.php?id=2547</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=2547</guid>
      <pubDate>Wed, 01 Jan 2025 00:00:00 +0000</pubDate>
      <category>effect:harm</category>
      <category>evidence:low</category>
      <category>year:2025</category>
      <category>study_type:animal</category>
      <description><![CDATA[<p><strong>Effect:</strong> Harm &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> animal</p>
<p><small>Neurotoxicology · 2025 · Manual</small></p>
<p>In vivo pre- and post-natal exposure at 0.08 and 0.4 W/kg SAR was associated with decreased BDNF levels, fewer BrdU+ proliferative cells, and a decreased excitatory/inhibitory synapse balance in rat pups. In vitro exposure at 0.08 W/kg SAR was associated with increased Ki-67+ proliferative cells, increased apoptosis, increased double-strand DNA breaks, and shifts in NSC-derived cell-type ratios (lower B1 cells; higher oligodendrocyte progenitor cells and astrocytes).</p>
<p><small>DOI: 10.1016/j.neuro.2025.103312</small></p>
<p><small><strong>Study URL:</strong> <a href="https://www.sciencedirect.com/science/article/pii/S0161813X2500110X?via%3Dihub" target="_blank" rel="noopener">https://www.sciencedirect.com/science/article/pii/S0161813X2500110X?via%3Dihub</a></small></p>]]></description>
    </item>
    <item>
      <title>Association between electromagnetic field exposure and abortion in pregnant women living in Tehran</title>
      <link>https://rfsafe.org/mel/paper.php?id=6757</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=6757</guid>
      <pubDate>Fri, 01 Jan 2016 00:00:00 +0000</pubDate>
      <category>effect:mixed</category>
      <category>evidence:low</category>
      <category>year:2016</category>
      <category>study_type:cohort</category>
      <description><![CDATA[<p><strong>Effect:</strong> Mixed &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> cohort</p>
<p><small>Int J Reprod Biomed · 2016 · Manual</small></p>
<p>Women exposed to higher measured EMF levels had a statistically significant increased likelihood of miscarriage by likelihood ratio test, but this association was not confirmed by the Wald test; overall miscarriage rate was 12.3%.</p>
<p><small>PMID: 27326421</small></p>
<p><small><strong>Study URL:</strong> <a href="https://pubmed.ncbi.nlm.nih.gov/27326421/" target="_blank" rel="noopener">https://pubmed.ncbi.nlm.nih.gov/27326421/</a></small></p>]]></description>
    </item>
    <item>
      <title>Immunohistochemical, Biochemical and Genetic Evaluation of the Effects of Ginseng Administration on Blood–Brain Barrier in Rats Exposed to 2100 MHz and 2450 MHz Electromagnetic Radiation</title>
      <link>https://rfsafe.org/mel/paper.php?id=6760</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=6760</guid>
      <pubDate>Fri, 27 Feb 2026 00:00:00 +0000</pubDate>
      <category>effect:mixed</category>
      <category>evidence:low</category>
      <category>year:2026</category>
      <category>study_type:animal</category>
      <description><![CDATA[<p><strong>Effect:</strong> Mixed &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> animal</p>
<p><small>Appl. Sci. · 2026 · Manual</small></p>
<p>Rats exposed to 2100 MHz or 2450 MHz EMF (1 h/day for 30 days) showed immunohistochemical evidence of apoptosis in brain tissue, with decreased COX-2 gene expression and increased BAX protein in EMF groups. Ginseng administration (150 mg/kg/day for 30 days) in exposed groups was concluded to reduce harmful effects by increasing COX-2 gene expression and decreasing BAX protein during the apoptosis process.</p>
<p><small>DOI: 10.3390/app16052376</small></p>
<p><small><strong>Study URL:</strong> <a href="https://doi.org/10.3390/app16052376" target="_blank" rel="noopener">https://doi.org/10.3390/app16052376</a></small></p>]]></description>
    </item>
    <item>
      <title>Numerical dosimetry dedicated to children RF exposure</title>
      <link>https://rfsafe.org/mel/paper.php?id=6751</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=6751</guid>
      <pubDate>Sat, 01 Jan 2011 00:00:00 +0000</pubDate>
      <category>effect:mixed</category>
      <category>evidence:low</category>
      <category>year:2011</category>
      <category>study_type:review</category>
      <description><![CDATA[<p><strong>Effect:</strong> Mixed &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> review</p>
<p><small>Prog Biophys Mol Biol · 2011 · Manual</small></p>
<p>The paper reports numerical dosimetry results indicating that current phone compliance methods are valid for children, while some tissues (e.g., peripheral brain tissues) may have higher exposure in children than adults. For plane-wave exposures, whole-body SAR in children can be higher than in adults, and compliance with ICNIRP reference levels may not guarantee compliance with ICNIRP basic restrictions. Preliminary foetus-model results suggest foetus exposure is often lower than the mother’s, with foetus position in the uterus influencing exposure.</p>
<p><small>DOI: 10.1016/j.pbiomolbio.2011.10.002 · PMID: 22005525</small></p>
<p><small><strong>Study URL:</strong> <a href="https://pubmed.ncbi.nlm.nih.gov/22005525/" target="_blank" rel="noopener">https://pubmed.ncbi.nlm.nih.gov/22005525/</a></small></p>]]></description>
    </item>
    <item>
      <title>Effects of industrial, scientific, and medical (ISM) band frequency 2.45 GHz on membrane integrity and oxidative stress of human skin bacteria</title>
      <link>https://rfsafe.org/mel/paper.php?id=6781</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=6781</guid>
      <pubDate>Thu, 26 Feb 2026 00:00:00 +0000</pubDate>
      <category>effect:harm</category>
      <category>evidence:low</category>
      <category>year:2026</category>
      <category>study_type:in_vitro</category>
      <description><![CDATA[<p><strong>Effect:</strong> Harm &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> in_vitro</p>
<p><small>Int J Radiat Biol · 2026 · Manual</small></p>
<p>Bacterial cultures exposed to 2.45 GHz RFR showed significantly increased ROS generation (~2-fold increases in total intracellular ROS, hydroxyl radicals, and superoxide radicals) versus sham/control. SEM/TEM indicated disrupted membranes and cytoplasmic disorganization, accompanied by increased protein/carbohydrate leakage and elevated oxidative damage markers (MDA &gt;1.5-fold; protein carbonyl &gt;2-fold), with effects reported across all three species and more pronounced damage in E. cloacae.</p>
<p><small>DOI: 10.1080/09553002.2026.2636305 · PMID: 41747184</small></p>
<p><small><strong>Study URL:</strong> <a href="https://pubmed.ncbi.nlm.nih.gov/41747184/" target="_blank" rel="noopener">https://pubmed.ncbi.nlm.nih.gov/41747184/</a></small></p>]]></description>
    </item>
    <item>
      <title>Male Reproductive and Cellular Damage After Prenatal 3.5 GHz Radiation Exposure: One-Year Postnatal Effects</title>
      <link>https://rfsafe.org/mel/paper.php?id=2506</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=2506</guid>
      <pubDate>Wed, 01 Jan 2025 00:00:00 +0000</pubDate>
      <category>effect:harm</category>
      <category>evidence:low</category>
      <category>year:2025</category>
      <category>study_type:animal</category>
      <description><![CDATA[<p><strong>Effect:</strong> Harm &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> animal</p>
<p><small>Ann N Y Acad Sci · 2025 · Manual</small></p>
<p>Compared with sham controls, prenatal 3.5 GHz RFR exposure was associated with reduced seminiferous tubule diameter and epithelial height and lower Johnsen scores in the full-gestation exposure group, and increased abnormal sperm morphology. Both exposure groups showed higher γ-H2AX immunostaining, increased TUNEL-positive cells, and higher apoptosis index; Beclin-1 expression was higher in the full-gestation exposure group.</p>
<p><small>DOI: 10.1111/nyas.70116</small></p>
<p><small><strong>Study URL:</strong> <a href="https://nyaspubs.onlinelibrary.wiley.com/doi/10.1111/nyas.70116" target="_blank" rel="noopener">https://nyaspubs.onlinelibrary.wiley.com/doi/10.1111/nyas.70116</a></small></p>]]></description>
    </item>
    <item>
      <title>Nanosecond electric pulse-induced calcium entry into chromaffin cells</title>
      <link>https://rfsafe.org/mel/paper.php?id=6745</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=6745</guid>
      <pubDate>Tue, 01 Jan 2008 00:00:00 +0000</pubDate>
      <category>effect:unclear</category>
      <category>evidence:low</category>
      <category>year:2008</category>
      <category>study_type:in_vitro</category>
      <description><![CDATA[<p><strong>Effect:</strong> Unclear &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> in_vitro</p>
<p><small>Bioelectrochemistry · 2008 · Manual</small></p>
<p>Chromaffin cells exposed to nanosecond electric pulses (2–8 MV/m) showed a rapid, short-lived increase in intracellular calcium after a single 4 ns, 8 MV/m pulse, with response magnitude dependent on field intensity. The calcium rise was unaffected by depletion of intracellular stores (caffeine or thapsigargin) but was prevented by extracellular EGTA, Co2+, or the L-type calcium channel blocker nitrendipine, consistent with calcium entry via L-type channels.</p>
<p><small>DOI: 10.1016/j.bioelechem.2008.02.003 · PMID: 18407807</small></p>
<p><small><strong>Study URL:</strong> <a href="https://pubmed.ncbi.nlm.nih.gov/18407807/" target="_blank" rel="noopener">https://pubmed.ncbi.nlm.nih.gov/18407807/</a></small></p>]]></description>
    </item>
    <item>
      <title>Treating cancer with amplitude-modulated electromagnetic fields: a potential paradigm shift, again?</title>
      <link>https://rfsafe.org/mel/paper.php?id=6742</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=6742</guid>
      <pubDate>Sun, 01 Jan 2012 00:00:00 +0000</pubDate>
      <category>effect:benefit</category>
      <category>evidence:low</category>
      <category>year:2012</category>
      <category>study_type:other</category>
      <description><![CDATA[<p><strong>Effect:</strong> Benefit &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> other</p>
<p><small>2012 · Manual</small></p>
<p>Amplitude-modulated electromagnetic fields (AM-EMF) exposure reduced growth rate of tumor cells from tissue-specific origins without affecting normal cells, with effects dependent on field strength and exposure time. Clinical reports indicated disease stabilization and partial responses in cancer patients treated with specific AM-EMF frequencies. The study suggests a biological mechanism involving gene expression changes and mitotic spindle dysfunction.</p>
<p><small><strong>Study URL:</strong> <a href="https://www.nature.com/articles/bjc2011576" target="_blank" rel="noopener">https://www.nature.com/articles/bjc2011576</a></small></p>]]></description>
    </item>
    <item>
      <title>Safety and Efficacy of amplitude-modulated radiofrequency electromagnetic fields in advanced hepatocellular carcinoma</title>
      <link>https://rfsafe.org/mel/paper.php?id=5709</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=5709</guid>
      <pubDate>Fri, 01 Jan 2021 00:00:00 +0000</pubDate>
      <category>effect:benefit</category>
      <category>evidence:low</category>
      <category>year:2021</category>
      <category>study_type:other</category>
      <description><![CDATA[<p><strong>Effect:</strong> Benefit &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> other</p>
<p><small>4Open · 2021 · Manual</small></p>
<p>In pooled patients treated with the TheraBionic P1 device, median OS was 10.36 months (Child-Pugh A, n=32), 4.44 months (Child-Pugh B, n=25), and 1.99 months (Child-Pugh C, n=2). Median OS for Child-Pugh A patients was 2.62 months longer than comparable historical controls (p=0.036); Child-Pugh B first-line therapy OS was slightly higher than historical controls receiving sorafenib. Reported adverse events were limited to grade 1 mucositis and fatigue, and no patients discontinued due to adverse events.</p>
<p><small>DOI: 10.1051/fopen/2021003 · PMID: 41550248</small></p>
<p><small><strong>Study URL:</strong> <a href="https://pubmed.ncbi.nlm.nih.gov/41550248/" target="_blank" rel="noopener">https://pubmed.ncbi.nlm.nih.gov/41550248/</a></small></p>]]></description>
    </item>
    <item>
      <title>Acceleration of the meckel syndrome by near-infrared light therapy</title>
      <link>https://rfsafe.org/mel/paper.php?id=6734</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=6734</guid>
      <pubDate>Sat, 01 Jan 2011 00:00:00 +0000</pubDate>
      <category>effect:harm</category>
      <category>evidence:low</category>
      <category>year:2011</category>
      <category>study_type:animal</category>
      <description><![CDATA[<p><strong>Effect:</strong> Harm &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> animal</p>
<p><small>Nephron Extra · 2011 · Manual</small></p>
<p>In cystic rats, near-infrared/red LED treatment (640–690 nm) reduced body weight and total kidney weight compared with sham-treated cystic rats, and BUN increased further (reported as almost 2-fold) with NIR treatment in both non-cystic and cystic rats. Hydrocephalus severity was reported as more severe in NIR-treated cystic rats, and the authors conclude that phototherapy initiated after symptom onset was detrimental to MKS-induced pathology.</p>
<p><small>DOI: 10.1159/000332046 · PMID: 22470396</small></p>
<p><small><strong>Study URL:</strong> <a href="https://pubmed.ncbi.nlm.nih.gov/22470396/" target="_blank" rel="noopener">https://pubmed.ncbi.nlm.nih.gov/22470396/</a></small></p>]]></description>
    </item>
    <item>
      <title>Risks of carcinogenesis from electromagnetic radiation of mobile telephony devices</title>
      <link>https://rfsafe.org/mel/paper.php?id=6732</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=6732</guid>
      <pubDate>Fri, 01 Jan 2010 00:00:00 +0000</pubDate>
      <category>effect:harm</category>
      <category>evidence:low</category>
      <category>year:2010</category>
      <category>study_type:review</category>
      <description><![CDATA[<p><strong>Effect:</strong> Harm &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> review</p>
<p><small>Exp Oncol · 2010 · Manual</small></p>
<p>The article summarizes epidemiological data reporting increased risk of certain tumors in long-term (over 10 years) mobile phone users, particularly with ipsilateral use (reported case-control odds ratios 1.3 to 6.1). It also notes two epidemiological studies reporting increased cancer incidence among people living close to mobile telephony base stations compared with more distant populations, and cites experimental findings of non-thermal biological effects (e.g., ROS, heat shock proteins, DNA damage, apoptosis).</p>
<p><small>PMID: 20693976</small></p>
<p><small><strong>Study URL:</strong> <a href="https://pubmed.ncbi.nlm.nih.gov/20693976/" target="_blank" rel="noopener">https://pubmed.ncbi.nlm.nih.gov/20693976/</a></small></p>]]></description>
    </item>
    <item>
      <title>Long-term exposure to microwave radiation provokes cancer growth: evidences from radars and mobile communication systems</title>
      <link>https://rfsafe.org/mel/paper.php?id=6731</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=6731</guid>
      <pubDate>Sat, 01 Jan 2011 00:00:00 +0000</pubDate>
      <category>effect:harm</category>
      <category>evidence:low</category>
      <category>year:2011</category>
      <category>study_type:review</category>
      <description><![CDATA[<p><strong>Effect:</strong> Harm &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> review</p>
<p><small>Exp Oncol · 2011 · Manual</small></p>
<p>The review describes epidemiological and experimental reports suggesting that long-term exposure to low-intensity microwave radiation can induce or promote cancer in humans and animal models, with effects typically appearing after long durations (up to 10+ years) and increased carcinogenesis reported in rodents after 17–24 months of exposure. It also cites reported metabolic stress-related changes (e.g., ROS, 8-hydroxi-2-deoxyguanosine, ornithine decarboxylase activation) and argues that current thermal-based safety guidelines (including ICNIRP) require reevaluation and that precautionary regulation is warranted.</p>
<p><small>PMID: 21716201</small></p>
<p><small><strong>Study URL:</strong> <a href="https://pubmed.ncbi.nlm.nih.gov/21716201/" target="_blank" rel="noopener">https://pubmed.ncbi.nlm.nih.gov/21716201/</a></small></p>]]></description>
    </item>
    <item>
      <title>Metabolic changes in cells under electromagnetic radiation of mobile communication systems</title>
      <link>https://rfsafe.org/mel/paper.php?id=6730</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=6730</guid>
      <pubDate>Sat, 01 Jan 2011 00:00:00 +0000</pubDate>
      <category>effect:harm</category>
      <category>evidence:low</category>
      <category>year:2011</category>
      <category>study_type:review</category>
      <description><![CDATA[<p><strong>Effect:</strong> Harm &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> review</p>
<p><small>Ukr Biokhim Zh (1999) · 2011 · Manual</small></p>
<p>This review summarizes recent research suggesting potential risks from long-term low-level microwave exposure from mobile communication systems and describes reported cellular/metabolic stress responses. Reproducible effects described include heat shock protein overexpression, increased ROS and intracellular Ca2+, DNA damage with inhibited repair, and apoptosis, with ERK and p38MAPK implicated; the authors argue effects are not exclusively thermal and suggest standards based only on thermal effects may need re-evaluation.</p>
<p><small>PMID: 21851043</small></p>
<p><small><strong>Study URL:</strong> <a href="https://pubmed.ncbi.nlm.nih.gov/21851043/" target="_blank" rel="noopener">https://pubmed.ncbi.nlm.nih.gov/21851043/</a></small></p>]]></description>
    </item>
    <item>
      <title>Changes in mitochondrial functioning with electromagnetic radiation of ultra high frequency as revealed by electron paramagnetic resonance methods</title>
      <link>https://rfsafe.org/mel/paper.php?id=6729</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=6729</guid>
      <pubDate>Wed, 01 Jan 2014 00:00:00 +0000</pubDate>
      <category>effect:harm</category>
      <category>evidence:low</category>
      <category>year:2014</category>
      <category>study_type:animal</category>
      <description><![CDATA[<p><strong>Effect:</strong> Harm &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> animal</p>
<p><small>Int J Radiat Biol · 2014 · Manual</small></p>
<p>In liver, cardiac, and aorta tissues from male rats exposed to non-thermal UHF EMR (pulsed and continuous), qualitative and quantitative disturbances in the mitochondrial ETC were reported, including iron-nitrosyl complex formation, decreased activity of FeS-protein N2, increased flavo-ubisemiquinone, and increased superoxide production rates. Effects in liver and aorta were reported as more pronounced with pulsed-mode exposure.</p>
<p><small>DOI: 10.3109/09553002.2014.899448 · PMID: 24597749</small></p>
<p><small><strong>Study URL:</strong> <a href="https://pubmed.ncbi.nlm.nih.gov/24597749/" target="_blank" rel="noopener">https://pubmed.ncbi.nlm.nih.gov/24597749/</a></small></p>]]></description>
    </item>
    <item>
      <title>GSM 900 MHz cellular phone radiation can either stimulate or depress early embryogenesis in Japanese quails depending on the duration of exposure</title>
      <link>https://rfsafe.org/mel/paper.php?id=6727</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=6727</guid>
      <pubDate>Tue, 01 Jan 2013 00:00:00 +0000</pubDate>
      <category>effect:mixed</category>
      <category>evidence:low</category>
      <category>year:2013</category>
      <category>study_type:animal</category>
      <description><![CDATA[<p><strong>Effect:</strong> Mixed &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> animal</p>
<p><small>Int J Radiat Biol · 2013 · Manual</small></p>
<p>GSM 900 MHz cellular phone radiation significantly altered embryonic development and DNA damage in a duration-dependent manner. After 38 h exposure, embryos had increased somite number (p&lt;0.001) and decreased DNA strand breaks (p&lt;0.001) versus control; after 158 h exposure, embryos had decreased somite number (p&lt;0.05) and increased DNA damage (p&lt;0.001) versus control.</p>
<p><small>DOI: 10.3109/09553002.2013.791408 · PMID: 23578013</small></p>
<p><small><strong>Study URL:</strong> <a href="https://pubmed.ncbi.nlm.nih.gov/23578013/" target="_blank" rel="noopener">https://pubmed.ncbi.nlm.nih.gov/23578013/</a></small></p>]]></description>
    </item>
    <item>
      <title>Overproduction of free radical species in embryonal cells exposed to low intensity radiofrequency radiation</title>
      <link>https://rfsafe.org/mel/paper.php?id=6726</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=6726</guid>
      <pubDate>Tue, 01 Jan 2013 00:00:00 +0000</pubDate>
      <category>effect:harm</category>
      <category>evidence:low</category>
      <category>year:2013</category>
      <category>study_type:animal</category>
      <description><![CDATA[<p><strong>Effect:</strong> Harm &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> animal</p>
<p><small>Exp Oncol · 2013 · Manual</small></p>
<p>RF-EMR exposure was associated with significant persistent overproduction of superoxide and nitrogen oxide across all analysis timepoints. Exposed embryos also showed significantly increased TBARS and 8-oxo-dG and significantly decreased superoxide dismutase and catalase activities compared with unexposed embryos.</p>
<p><small>PMID: 24084462</small></p>
<p><small><strong>Study URL:</strong> <a href="https://pubmed.ncbi.nlm.nih.gov/24084462/" target="_blank" rel="noopener">https://pubmed.ncbi.nlm.nih.gov/24084462/</a></small></p>]]></description>
    </item>
    <item>
      <title>Oxidative and mutagenic effects of low intensity GSM 1800 MHz microwave radiation</title>
      <link>https://rfsafe.org/mel/paper.php?id=6725</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=6725</guid>
      <pubDate>Mon, 01 Jan 2018 00:00:00 +0000</pubDate>
      <category>effect:harm</category>
      <category>evidence:low</category>
      <category>year:2018</category>
      <category>study_type:animal</category>
      <description><![CDATA[<p><strong>Effect:</strong> Harm &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> animal</p>
<p><small>Exp Oncol · 2018 · Manual</small></p>
<p>Quail embryos exposed in ovo to low-intensity GSM 1800 MHz radiation (0.32 µW/cm2; 48 s on/12 s off) showed statistically significant increases in oxidative markers (2-fold higher superoxide generation; 85% higher nitrogen oxide generation), DNA integrity damage, and increased oxidative DNA damage (up to ~2x higher 8-oxo-dG in cells of 1-day-old chicks). Exposure was also associated with an almost twofold increase in embryo mortality.</p>
<p><small>PMID: 30593748</small></p>
<p><small><strong>Study URL:</strong> <a href="https://pubmed.ncbi.nlm.nih.gov/30593748/" target="_blank" rel="noopener">https://pubmed.ncbi.nlm.nih.gov/30593748/</a></small></p>]]></description>
    </item>
    <item>
      <title>Electromagnetic fields act via activation of voltage-gated calcium channels to produce beneficial or adverse effects</title>
      <link>https://rfsafe.org/mel/paper.php?id=2424</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=2424</guid>
      <pubDate>Tue, 01 Jan 2013 00:00:00 +0000</pubDate>
      <category>effect:mixed</category>
      <category>evidence:low</category>
      <category>year:2013</category>
      <category>study_type:review</category>
      <description><![CDATA[<p><strong>Effect:</strong> Mixed &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> review</p>
<p><small>J Cell Mol Med · 2013 · Manual</small></p>
<p>This review summarizes literature suggesting that non-thermal EMF effects (ELF and microwave range) can be reduced or blocked by L-type or other VGCC blockers, supporting VGCCs as direct targets. It proposes downstream signaling via Ca2+/calmodulin-dependent nitric oxide pathways that may underlie both therapeutic effects (e.g., bone growth stimulation) and adverse effects (e.g., DNA single-strand breaks via oxidative stress/peroxynitrite).</p>
<p><small>DOI: 10.1111/jcmm.12088 · PMID: 23802593</small></p>
<p><small><strong>Study URL:</strong> <a href="https://pmc.ncbi.nlm.nih.gov/articles/PMC3780531/" target="_blank" rel="noopener">https://pmc.ncbi.nlm.nih.gov/articles/PMC3780531/</a></small></p>]]></description>
    </item>
    <item>
      <title>Real versus Simulated Mobile Phone Exposures in Experimental Studies</title>
      <link>https://rfsafe.org/mel/paper.php?id=6723</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=6723</guid>
      <pubDate>Thu, 01 Jan 2015 00:00:00 +0000</pubDate>
      <category>effect:harm</category>
      <category>evidence:low</category>
      <category>year:2015</category>
      <category>study_type:review</category>
      <description><![CDATA[<p><strong>Effect:</strong> Harm &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> review</p>
<p><small>Biomed Res Int · 2015 · Manual</small></p>
<p>The authors argue that real-life emissions from commercially available mobile phones are variable and differ substantially from simulated, fixed-parameter emissions used in many experiments. They state that experimental studies using simulated emissions show strong inconsistency (with less than 50% reporting effects), whereas studies using real mobile phone exposures show nearly 100% consistency in reporting adverse effects, and they conclude experiments should use commercially available handsets to better reflect real-world exposure.</p>
<p><small>DOI: 10.1155/2015/607053 · PMID: 26346766</small></p>
<p><small><strong>Study URL:</strong> <a href="https://pubmed.ncbi.nlm.nih.gov/26346766/" target="_blank" rel="noopener">https://pubmed.ncbi.nlm.nih.gov/26346766/</a></small></p>]]></description>
    </item>
    <item>
      <title>Comparing DNA damage induced by mobile telephony and other types of man-made electromagnetic fields</title>
      <link>https://rfsafe.org/mel/paper.php?id=1859</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=1859</guid>
      <pubDate>Tue, 01 Jan 2019 00:00:00 +0000</pubDate>
      <category>effect:harm</category>
      <category>evidence:low</category>
      <category>year:2019</category>
      <category>study_type:review</category>
      <description><![CDATA[<p><strong>Effect:</strong> Harm &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> review</p>
<p><small>Mutat Res Rev Mutat Res · 2019 · Manual</small></p>
<p>This review summarizes results from the author’s group (2006–2016) comparing DNA fragmentation in Drosophila melanogaster oogenesis under identical procedures after exposure to six different EMFs. It reports that real-life mobile telephony (mobile phone) EMFs were significantly more bioactive/damaging than a 50 Hz alternating magnetic field or a pulsed electric field, even with shorter exposure durations, and were described as more damaging than certain previously tested cytotoxic stressors (e.g., certain chemicals, starvation, dehydration).</p>
<p><small>DOI: 10.1016/j.mrrev.2019.03.003</small></p>
<p><small><strong>Study URL:</strong> <a href="https://www.sciencedirect.com/science/article/pii/S1383574218300991" target="_blank" rel="noopener">https://www.sciencedirect.com/science/article/pii/S1383574218300991</a></small></p>]]></description>
    </item>
    <item>
      <title>One or many environmental intolerance(s)? A cluster analysis over two representative samples</title>
      <link>https://rfsafe.org/mel/paper.php?id=6716</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=6716</guid>
      <pubDate>Thu, 01 Jan 2026 00:00:00 +0000</pubDate>
      <category>effect:unclear</category>
      <category>evidence:low</category>
      <category>year:2026</category>
      <category>study_type:cross_sectional</category>
      <description><![CDATA[<p><strong>Effect:</strong> Unclear &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> cross_sectional</p>
<p><small>International Journal of Hygiene and Environmental Health · 2026 · Manual</small></p>
<p>Across two representative datasets, cluster analysis distinguished people with versus without SAEFs, but did not find evidence for empirically distinct SAEF subtype clusters. The SAEF cluster showed higher chemical, noise, and electromagnetic field sensitivity and worse sleep, along with higher markers of psychopathology (e.g., anxiety, depression), and included more women.</p>
<p><small>DOI: 10.1016/j.ijheh.2026.114764</small></p>
<p><small><strong>Study URL:</strong> <a href="https://doi.org/10.1016/j.ijheh.2026.114764" target="_blank" rel="noopener">https://doi.org/10.1016/j.ijheh.2026.114764</a></small></p>]]></description>
    </item>
    <item>
      <title>Overlap in prevalence between various types of environmental intolerance</title>
      <link>https://rfsafe.org/mel/paper.php?id=6715</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=6715</guid>
      <pubDate>Wed, 01 Jan 2014 00:00:00 +0000</pubDate>
      <category>effect:unclear</category>
      <category>evidence:low</category>
      <category>year:2014</category>
      <category>study_type:cross_sectional</category>
      <description><![CDATA[<p><strong>Effect:</strong> Unclear &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> cross_sectional</p>
<p><small>Int J Hyg Environ Health · 2014 · Manual</small></p>
<p>Using questionnaire-based cross-sectional data, overlaps between the four environmental intolerances were greater than predicted by coincidence for both self-reported and diagnosed cases, except for the overlap between diagnosed intolerance to sounds and EMFs.</p>
<p><small>DOI: 10.1016/j.ijheh.2013.08.005 · PMID: 24029726</small></p>
<p><small><strong>Study URL:</strong> <a href="https://pubmed.ncbi.nlm.nih.gov/24029726/" target="_blank" rel="noopener">https://pubmed.ncbi.nlm.nih.gov/24029726/</a></small></p>]]></description>
    </item>
    <item>
      <title>Non-thermal biological effects of radiofrequency electromagnetic radiation: Mechanistic insights into male reproductive vulnerability in the era of ubiquitous exposure</title>
      <link>https://rfsafe.org/mel/paper.php?id=2498</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=2498</guid>
      <pubDate>Wed, 01 Jan 2025 00:00:00 +0000</pubDate>
      <category>effect:harm</category>
      <category>evidence:low</category>
      <category>year:2025</category>
      <category>study_type:review</category>
      <description><![CDATA[<p><strong>Effect:</strong> Harm &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> review</p>
<p><small>Reprod Toxicol · 2025 · Manual</small></p>
<p>This review summarizes mechanistic pathways by which chronic low-intensity RF-EMR exposure may impair male reproductive health, emphasizing oxidative stress/redox imbalance, mitochondrial dysfunction, DNA damage, apoptosis, autophagy dysregulation, and hormonal disruption via HPG axis interference. It states that experimental and clinical studies consistently link RF-EMR exposure to reduced sperm count and motility, altered testicular architecture, blood-testis barrier compromise, and suppressed testosterone synthesis, including at SARs below current safety thresholds, and notes emerging evidence for epigenetic/transgenerational effects and knowledge gaps for 5G/mmWave.</p>
<p><small>DOI: 10.1016/j.reprotox.2025.109087</small></p>
<p><small><strong>Study URL:</strong> <a href="https://www.sciencedirect.com/science/article/pii/S0890623825002588" target="_blank" rel="noopener">https://www.sciencedirect.com/science/article/pii/S0890623825002588</a></small></p>]]></description>
    </item>
    <item>
      <title>From adults to offspring: Wi-Fi RF-EMR exposure in adult zebrafish impairs reproduction and transgenerationally effects development and behavior of progeny</title>
      <link>https://rfsafe.org/mel/paper.php?id=2494</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=2494</guid>
      <pubDate>Wed, 01 Jan 2025 00:00:00 +0000</pubDate>
      <category>effect:harm</category>
      <category>evidence:low</category>
      <category>year:2025</category>
      <category>study_type:animal</category>
      <description><![CDATA[<p><strong>Effect:</strong> Harm &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> animal</p>
<p><small>Sci Total Environ · 2025 · Manual</small></p>
<p>Adult zebrafish exposed to Wi‑Fi RF‑EMR (4 h/day for 30 days) showed significant gonadal histopathology, including testicular abnormalities (seminiferous tubule disintegration, reduced spermatids, abnormal lumina) and ovarian follicular degeneration/stromal disruption. Progeny of exposed adults (raised EMF-free) had increased mortality and gross malformations, with malformation rates increasing with longer parental exposure, and larvae showed anxiety-like behavior at 10 dpf.</p>
<p><small>DOI: 10.1016/j.scitotenv.2025.180982 · PMID: 41264945</small></p>
<p><small><strong>Study URL:</strong> <a href="https://pubmed.ncbi.nlm.nih.gov/41264945/" target="_blank" rel="noopener">https://pubmed.ncbi.nlm.nih.gov/41264945/</a></small></p>]]></description>
    </item>
    <item>
      <title>Transgenerational changes in Daphnia magna under radio frequency radiation in the juvenile and puberty period</title>
      <link>https://rfsafe.org/mel/paper.php?id=781</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=781</guid>
      <pubDate>Sun, 01 Jan 2023 00:00:00 +0000</pubDate>
      <category>effect:harm</category>
      <category>evidence:low</category>
      <category>year:2023</category>
      <category>study_type:animal</category>
      <description><![CDATA[<p><strong>Effect:</strong> Harm &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> animal</p>
<p><small>Int J Radiat Biol · 2023 · Manual</small></p>
<p>Chronic RF-EMF exposure during the juvenile period significantly affected fertility and size of exposed D. magna and their first-generation offspring. Decreased fertility was suggested to be associated with cytotoxic effects in irradiated animals, and reduced terminal spine/body size indicated negative impacts on protective strategy. Reproductive parameters were reported to be restored by the second generation.</p>
<p><small>DOI: 10.1080/09553002.2022.2087928 · PMID: 35675553</small></p>
<p><small><strong>Study URL:</strong> <a href="https://pubmed.ncbi.nlm.nih.gov/35675553/" target="_blank" rel="noopener">https://pubmed.ncbi.nlm.nih.gov/35675553/</a></small></p>]]></description>
    </item>
    <item>
      <title>Multigenerational effects of whole body exposure to 2.14 GHz W-CDMA cellular phone signals on brain function in rats</title>
      <link>https://rfsafe.org/mel/paper.php?id=4663</link>
      <guid isPermaLink="true">https://rfsafe.org/mel/paper.php?id=4663</guid>
      <pubDate>Wed, 01 Jan 2014 00:00:00 +0000</pubDate>
      <category>effect:no_effect</category>
      <category>evidence:low</category>
      <category>year:2014</category>
      <category>study_type:animal</category>
      <description><![CDATA[<p><strong>Effect:</strong> No effect &nbsp; <strong>Evidence:</strong> Low</p>
<p><strong>Study type:</strong> animal</p>
<p><small>Bioelectromagnetics · 2014 · Manual</small></p>
<p>Across three generations of rats exposed whole-body to 2.14 GHz W-CDMA signals for 20 h/day at designed average SAR levels high (&lt;0.24 W/kg), low (&lt;0.08 W/kg), or sham, no abnormalities were observed in mothers or offspring in measured biological parameters, including neurobehavioral function. The authors concluded no adverse effects on F1–F3 offspring under these experimental conditions.</p>
<p><small>DOI: 10.1002/bem.21871 · PMID: 25196377</small></p>
<p><small><strong>Study URL:</strong> <a href="https://pubmed.ncbi.nlm.nih.gov/25196377/" target="_blank" rel="noopener">https://pubmed.ncbi.nlm.nih.gov/25196377/</a></small></p>]]></description>
    </item>
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