Devolving One Calcium Burst at a Time
This RF Safe article by John Coates argues that “non-native” RF/ELF electromagnetic fields may degrade biological “signal fidelity” by perturbing voltage-gated ion channel timing, with downstream effects on mitochondria, reactive oxygen species (ROS), and redox biology. It presents a conceptual “S4–Mito–Spin” framework and cites selected studies and mechanisms (e.g., ion-channel forced oscillation, radical-pair/spin chemistry) to support the plausibility of non-thermal effects. The piece frames modern wireless infrastructure as an uncontrolled long-term experiment and suggests current regulation focuses too narrowly on heating.
Key points
- Claims that modern RF/ELF exposures introduce “timing noise” into bioelectric signaling, particularly via voltage-gated ion channels (S4 segments) in excitable tissues.
- Argues non-thermal mechanisms could matter, asserting that biologically relevant perturbations can occur without appreciable heating.
- Cites Panagopoulos’ work as support for an ion-channel forced oscillation (IFO-VGIC) mechanism affecting channel gating fidelity.
- References Durdík et al. (2019) as reporting increased RF-induced ROS in more differentiated human cord-blood cell populations under non-thermal UMTS exposure conditions.
- Extends the argument to spin-chemistry/radical-pair mechanisms (e.g., cryptochrome-based magnetoreception) as a possible pathway for RF/magnetic fields to influence redox balance.
- Frames the issue as a precautionary concern and criticizes regulatory approaches for focusing primarily on thermal effects.
Referenced studies & papers
Relevant papers in OpenMel
Source:
Open original
AI-generated summaries may be incomplete or incorrect. This content is for informational purposes only and is not medical advice.
AI-generated summaries may be incomplete or incorrect. This content is for informational purposes only and is not medical advice.
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