This preprint discusses the microwave auditory effect, in which pulsed microwave exposure can produce perceived clicks or buzzing sensations. It considers whether acoustic pressures in the head generated by pulsed microwaves could explain health conditions such as "Havana Syndrome." The abstract emphasizes evaluating potential risks from electromagnetic field exposures but does not provide specific methods or quantitative results.

This study used computational dosimetry to analyze induced electric fields in a realistic human body model for a 60 Hz magnetic-field exposure system targeting the leg. Simulations indicated high EF intensities in several leg nerves and modeled conditions consistent with possible peripheral nerve stimulation. The MRG model produced lower stimulation thresholds than the SENN model, and nerve orientation was reported as a key determinant of stimulation risk.

This animal ecology study reports that weak anthropogenic-like airborne electric fields can reduce honeybee floral landing rates. It reports deterrent effects for AC and positive DC fields, with no statistically significant effect for negative DC fields. The authors also report that electric fields measured near high-voltage power lines can match the levels that affected bees and extend tens of meters at foraging-relevant heights.

This laboratory study tested combined effects of constant temperature and RF exposure on development of Aedes aegypti and Aedes albopictus from hatching to adult emergence. Temperature was reported as the primary determinant of developmental timing, with optimal development around 302C. RF exposure (900 MHz and 18 GHz) was described as a secondary factor that could accelerate or prolong development depending on temperature, with synergistic shortening at 250C and prolongation under suboptimal conditions.

This animal study exposed zebrafish embryos to static electromagnetic fields for 63 days post-hatching across aquariums positioned 30–99 cm from the source, with an EMF-free control. The abstract reports strong shifts in sex distribution (including 100% female at the closest distance), markedly reduced survival in exposed groups, and histological liver and gonadal damage. The authors frame the findings as evidence of potential ecological risk via disrupted sex ratios and compromised health.

This prospective cohort study followed 105 neonates/infants for one year and measured household RF-EMF using a selective radiation meter, categorizing exposure into tertiles. Higher household RF-EMF exposure was associated with lower ASQ-3 neurodevelopmental scores, particularly in motor and problem-solving domains, and higher odds of monitor/refer classifications for fine motor and problem-solving. The abstract notes these associations persisted after adjustment for low birth weight, though exposure was measured at a single time point and key confounders (e.g., prenatal phone use, parental interaction) were not assessed.

This scoping review and evidence map (PRISMA-ScR) summarizes over 500 studies on RF-EMF exposure and genotoxicity across in vitro, in vivo, and epidemiological research. The authors report a higher proportion of significant DNA damage findings in in vivo and epidemiological studies than in vitro studies, with DNA base damage commonly reported under real-world/pulsed/GSM talk-mode conditions and longer exposures. They conclude that DNA damage has been observed at exposure levels below ICNIRP limits and recommend precautionary measures and updates to guidelines to address potential non-thermal effects.

This narrative article examines how opposing views formed regarding health and biological effects of electromagnetic radiation, focusing on ELF and RF exposures. It highlights historical controversies (e.g., childhood leukemia and ELF fields) and disputes over thermal versus non-thermal effects and reliance on SAR. The author argues that social and institutional factors, including industry influence, shaped interpretation and public discourse around EMF safety.

This case-control genetic association study in men from West Bengal, India examined variants in meiotic regulator genes (SPO11, RNF212, SYCP3) alongside reported exposure to electronic (electromagnetic) radiation as risk factors for azoospermia. It reports that genetic variants were associated with increased azoospermia risk, and that risk was higher among men aged 30+ who were also exposed to electromagnetic radiation. The authors conclude that EMF exposure may exacerbate fertility impairment in genetically predisposed men.