This PubMed-listed paper argues that the U.S. regulatory framework for radiofrequency radiation (RFR) from wireless technologies is outdated, lacks adequate oversight and enforcement, and has not been meaningfully updated since 1996. It contends that FCC exposure limits focus on short-term, high-intensity effects and do not address long-term, low-intensity exposures, with insufficient safeguards for children, pregnancy, and other vulnerable groups. The authors also discuss alleged regulatory capture, gaps in monitoring and compliance, and propose reforms including independent research, updated safety limits, and stronger pre- and post-market surveillance.

This RF Safe article argues that the organization’s EMF safety advocacy should not be dismissed as “biased” or “commercially motivated,” framing its work as rooted in its founder’s personal experience and long-term activism. It recounts founder John Coates’ claim that prenatal RF exposure contributed to his infant daughter’s neural tube defect, and presents RF Safe as combining advocacy, scientific synthesis, and product development. The piece also claims RF Safe’s antenna work helped prompt a 2003 FCC rule change recognizing directional antenna approaches to reduce energy toward users while maintaining performance.

This policy-focused paper contends that U.S. oversight of radiofrequency radiation from wireless technologies is outdated and insufficient, with exposure limits and testing approaches not aligned with modern long-term, chronic exposure scenarios. It emphasizes gaps in protections for children, pregnancy, vulnerable populations, workers, and wildlife, and describes limited monitoring, research, and enforcement capacity. The author proposes reforms to improve independent research, science-based limits, surveillance, and regulatory transparency.

This study uses anatomically detailed computational models of a five-year-old girl, a pregnant woman in the third trimester, and a fetus to simulate mobile phone RF exposure inside an elevator cabin. Simulations at 1000 MHz and 1800 MHz across 48 configurations evaluated SAR10g, whole-body SAR, and maximum temperature. The abstract reports that configuration (positioning and phone orientation) can substantially change absorption and temperature metrics and calls for broader scenario testing to inform safety guidance for vulnerable populations.

This animal study examined maternal mobile phone exposure during different gestational windows in Wistar rats and assessed ovarian development and hormones in neonatal offspring. Compared with sham (phone off), exposed groups (standby and conversation/standby) were reported to have lower neonatal estrogen and progesterone and reduced primordial follicle/primary oocyte measures, with stronger effects after longer exposure. The study also reports increased primordial follicle apoptosis, particularly in the conversation/standby condition, and notes effects even with first-week gestational exposure.

This animal study examined whether continuous prenatal exposure to 2450 MHz radiofrequency radiation (simulating Wi‑Fi) affects thyroid tissue in rat offspring at 6 months. The exposed group showed significantly increased mononuclear cell infiltration and vascular congestion in thyroid histology. TUNEL-positive cell percentage and H2A.X antibody levels did not differ significantly between groups.