This review discusses epidemiological and mechanistic reports linking EMF exposure with oxidative stress and disease risk, and introduces an Electromagnetic Pathogenesis (EMP) conceptual model. The model proposes that non-ionizing EMFs increase mitochondrial electron leakage via electron tunneling, raising free radical production and oxidative stress. The authors argue oxidative stress is a primary mechanism connecting EMF exposure to cancer, cardiovascular, neurodevelopmental/neurodegenerative diseases, and behavioral/reproductive effects, and suggest reducing exposure may lower risk.

This ex-vivo experimental study tested whether electromagnetic fields from a commercial hair dryer affect microleakage of class V dental amalgam restorations in 100 extracted human teeth. Several exposure groups showed significantly higher dye-penetration microleakage scores than the unexposed control, while one exposure condition did not differ from control. The authors conclude that hair-dryer EMF exposure can increase microleakage and potentially compromise restoration integrity.

This rat study tested whether acute exposure to 0.9 and 1.8 GHz continuous-wave radiofrequency radiation alters blood-brain barrier permeability. Using Evans-blue/albumin as a tracer, the authors report no BBB leakage in exposed female rats but a significant increase in albumin in exposed male rat brains versus sham. The authors interpret this as suggesting BBB/vascular permeability changes in males at SAR levels stated to be below international limits.

This exposure assessment measured 20 kHz magnetic field leakage from induction cooking heaters across four models and compared results with ICNIRP general public limits. The maximum reported magnetic flux density was 16 µT at a specified measurement point using two S-type pans. Field leakage depended on pan size and configuration, and finite element modeling was reported to align closely with measurements.