Archive

3 posts

Density‑Gated Spin Engines: Why the 5G Skin‑Cell Null Fits the Heme/Spin Extension

Independent Voices RF Safe Nov 24, 2025

This RF Safe commentary argues that non-thermal RF/5G effects may vary by tissue based on the density of specific biological “targets,” such as voltage-gated channel S4 helices, mitochondrial/NOX ROS capacity, and heme/flavin “spin chemistry” substrates. It claims that reported null findings in 5G mmWave skin-cell studies can be reconciled with reported red blood cell (RBC) rouleaux observations by proposing a “density-gated” mechanism where spin-related effects are more detectable in heme-dense cells like RBCs. The post cites an ultrasound study (named “Brown & Biebrich”) as showing in-vivo rouleaux changes within minutes near a smartphone, but provides limited methodological detail in the excerpt.

The RF Radiation Safety Story

Independent Voices RF Safe Nov 14, 2025

This RF Safe article argues that U.S. radiofrequency (RF) exposure policy is outdated, emphasizing that FCC limits adopted in 1996 are based on preventing tissue heating and do not address alleged non-thermal biological effects. It claims responsibility for protecting public health from electronic product radiation was effectively ceded from health agencies to the FCC, and that Section 704 of the Telecommunications Act limits local governments from opposing wireless infrastructure on health grounds if FCC limits are met. The piece cites epidemiology, cell studies, and animal studies (notably the U.S. National Toxicology Program and the Ramazzini Institute) to argue that evidence has accumulated and regulation should be updated, but it presents these points in an advocacy framing rather than as a balanced review.

What non‑native EMFs really do —the rise of immune‑driven disease

Independent Voices RF Safe Nov 5, 2025

This RF Safe article argues that “non-native” electromagnetic fields (from power systems, radio, and mobile/5G signals) can disrupt the timing of voltage-gated ion channel activity in immune cells, leading to altered immune signaling, mitochondrial stress, and chronic inflammation. It links these proposed mechanisms to increases in autoimmune-type and immune-driven diseases over time, and cites a mix of reviews, cell studies, animal studies, and rodent bioassays as supportive evidence. The piece frames EMF risk as driven by signal timing/patterning rather than heating, and calls for regulation and engineering changes to address these effects.

Page 1 / 1