RF Safe reports that HHS confirmed plans to launch a new study on cellphone radiation and that an HHS spokesperson said the FDA removed webpages with “old conclusions” while new research is undertaken to identify knowledge gaps, including for emerging technologies. The article frames the FDA webpage changes as a meaningful shift away from categorical reassurance, while noting Reuters’ reporting that some FDA and CDC pages still state there is no credible evidence of health problems from cellphone radiation. It also links the development to the 2021 D.C. Circuit decision in Environmental Health Trust v. FCC, arguing the ruling exposed weaknesses in the FCC’s reliance on other agencies’ statements.

RF Safe publishes a rebuttal to Media Bias Fact Check’s (MBFC) January 8, 2026 credibility assessment, arguing MBFC’s “Medium Credibility” rating is unjustified despite MBFC upgrades to “Least Biased” and “Mostly Factual.” The post disputes MBFC’s criticisms (selective citation, alarmist framing, and potential conflict of interest from product sales) and claims RF Safe’s coverage aligns with WHO-commissioned reviews and legal/regulatory developments. RF Safe reiterates its view that thermal-only RF exposure guidelines are inadequate and calls for policy reform while stating it does not claim definitive human causation.

RF Safe argues that celebrity-branded mobile services (citing reported plans for “Beast Mobile” and the announced “Trump Mobile”) could normalize near-body, all-day phone use—especially among children—and therefore carry ethical responsibility for scaled RF exposure. The piece cites legal and scientific developments (including the 2021 Environmental Health Trust v. FCC decision, the U.S. NTP animal studies, and a WHO-commissioned systematic review) to claim the evidence base has “moved decisively” toward concern about long-term RF-EMF effects. It also promotes a proposed mechanistic framework ("S4–Mito–Spin") and suggests shifting indoor connectivity toward Li‑Fi (IEEE 802.11bb) as a harm-reduction approach.

RF Safe recaps a Truth Expedition podcast episode featuring RF Safe founder John Coates discussing alleged biological risks from EMF exposure and arguing that current regulations lag behind modern science. The piece links EMFs to developmental and health concerns (including neural-tube defects and autism) via Coates’ proposed “S4–Mito–Spin” framework involving voltage-gated ion channels, mitochondrial signaling, and radical-pair/spin chemistry. It also promotes RF Safe’s research library, SAR comparison tools, and mitigation products as part of a risk-reduction approach.

This RF Safe article argues that the organization’s EMF safety advocacy should not be dismissed as “biased” or “commercially motivated,” framing its work as rooted in its founder’s personal experience and long-term activism. It recounts founder John Coates’ claim that prenatal RF exposure contributed to his infant daughter’s neural tube defect, and presents RF Safe as combining advocacy, scientific synthesis, and product development. The piece also claims RF Safe’s antenna work helped prompt a 2003 FCC rule change recognizing directional antenna approaches to reduce energy toward users while maintaining performance.

This RF Safe article defends the organization against accusations of bias, framing its EMF safety advocacy as rooted in founder John Coates’ personal tragedy and long-term efforts in product development, research synthesis, and policy reform. It claims RF Safe helped drive an FCC rule change related to antenna design and promotes various exposure-reduction accessories and training tools. The piece argues that non-thermal biological effects of RF/ELF fields are being overlooked by regulators and calls for policy changes such as revisiting Section 704 of the 1996 Telecom Act and shifting health oversight away from the FCC.

An RF Safe article presents a personal narrative and hypothesis that “non-native EMFs” act as “entropic waste” that could disrupt early embryonic neurodevelopment (neurulation), potentially contributing to neural-tube defects and later neurodevelopmental outcomes such as autism/ADHD. The author links a family tragedy to this hypothesis and argues for reducing wireless exposure as a precaution. The post cites several studies/reports (e.g., Farrell 1997, Aldad 2012, NTP 2018, WHO SR4A 2025) but does not provide detailed methods or evidence appraisal within the excerpt.

This RF Safe article argues that a critical embryonic window during neural tube formation (around days 21–28 post-conception) may link neural tube defects and a “major subset” of autism/ADHD-like traits, and it suggests electromagnetic fields could be a contributing factor. The author connects a personal story about a child’s death from a neural tube defect with claims about chick-embryo research reporting increased neural-tube malformations under weak electromagnetic fields. The piece also references developmental biology literature to support the broader idea that early embryogenesis/neurulation can influence later neurodevelopment, while presenting a speculative bioelectric/ion-channel mechanism.

RF Safe argues that a single biological mechanism explains widespread harm to children from modern wireless signals (cell phones, Wi‑Fi, 5G, DECT), emphasizing that these signals are “pulsed and modulated.” The post claims that “animal proof” is now high-certainty and references “WHO 2025 GRADE-rated systematic reviews,” linking EMF exposure to rare cancers in young people, declining sperm counts, and childhood autoimmune/neurodevelopmental disorders. The excerpt provided does not include citations or details sufficient to verify these claims.

RF Safe argues that current RF-EMF exposure standards are overly focused on thermal effects and should be replaced with a “post-thermal” regulatory paradigm that accounts for claimed non-thermal biological impacts. The piece cites a mix of mechanistic hypotheses, animal studies, epidemiology, and legal/policy developments (e.g., the 2021 D.C. Circuit EHT v. FCC decision) to support a precautionary reform agenda. It also asserts that recent WHO work in 2025 strengthens the case for tumor-related risks, though these characterizations are presented as the author’s interpretation rather than independently verified within the feed item.

This review argues that EMF exposure is associated in the literature with several adverse central nervous system outcomes, including blood-brain barrier disruption, oxidative stress, neurotransmitter changes, cognitive effects, and neurodevelopmental impacts. It reports that evidence on EMFs and brain tumors is conflicting, while noting WHO’s classification of radiofrequency EMFs as possibly carcinogenic to humans. The authors highlight prenatal and childhood periods as potentially more vulnerable and call for more standardized long-term and mechanistic research to guide public health policy.

This animal study exposed Aedes aegypti larvae to 5G-NR RF-EMF at 3.6 GHz for 5 days under two feeding regimes. The study reports delayed development at a lower exposure level mainly in nutritionally weakened larvae, and at a higher exposure level reports developmental changes and reduced adult size attributed to dielectric heating. Mortality and wing length asymmetry were reported as unchanged, and the authors note such high exposure levels are unlikely in natural aquatic settings.

This animal study examined Wi-Fi RF-EMR exposure in adult zebrafish (4 hours/day for 30 days) and assessed reproductive tissues and offspring outcomes. The abstract reports testicular and ovarian histopathological abnormalities in exposed adults. Offspring from exposed parents, maintained under EMF-free conditions, reportedly showed increased mortality, morphological abnormalities, and anxiety-like behavior, with malformations increasing with longer parental exposure.

This review discusses epidemiological and mechanistic reports linking EMF exposure with oxidative stress and disease risk, and introduces an Electromagnetic Pathogenesis (EMP) conceptual model. The model proposes that non-ionizing EMFs increase mitochondrial electron leakage via electron tunneling, raising free radical production and oxidative stress. The authors argue oxidative stress is a primary mechanism connecting EMF exposure to cancer, cardiovascular, neurodevelopmental/neurodegenerative diseases, and behavioral/reproductive effects, and suggest reducing exposure may lower risk.

This in vitro study reports that radiofrequency (RF) exposure in the 800–2,400 MHz range modulates differentiation pathways in human cortical organoids derived from embryonic stem cells. RF exposure is described as maintaining radial glia stem cell identity and delaying differentiation, alongside induction of endogenous retrovirus expression and increased expression of ASD-associated genes and retroelements. The abstract attributes these effects to dysregulation of BET proteins and reports that BET inhibition rescues the RF-associated developmental defects.

This animal and in vitro study examined non-thermal 900 MHz RF-EMF exposure during prenatal and postnatal development at 0.08 and 0.4 W/kg SAR. The authors report changes consistent with altered neurodevelopment, including reduced BDNF, reduced in vivo cell proliferation, and disrupted synaptic balance in rat pup brain regions. In vitro, exposed neural stem cells showed increased apoptosis and DNA double-strand breaks and shifts in cell populations toward glial lineages. The authors conclude that regulatory-level 900 MHz exposure can disrupt key neurodevelopmental processes in rodents.

This laboratory study tested combined effects of constant temperature and RF exposure on development of Aedes aegypti and Aedes albopictus from hatching to adult emergence. Temperature was reported as the primary determinant of developmental timing, with optimal development around 302C. RF exposure (900 MHz and 18 GHz) was described as a secondary factor that could accelerate or prolong development depending on temperature, with synergistic shortening at 250C and prolongation under suboptimal conditions.

This prospective cohort study followed 105 neonates/infants for one year and measured household RF-EMF using a selective radiation meter, categorizing exposure into tertiles. Higher household RF-EMF exposure was associated with lower ASQ-3 neurodevelopmental scores, particularly in motor and problem-solving domains, and higher odds of monitor/refer classifications for fine motor and problem-solving. The abstract notes these associations persisted after adjustment for low birth weight, though exposure was measured at a single time point and key confounders (e.g., prenatal phone use, parental interaction) were not assessed.

This animal study examined continual 2.4 GHz Wi‑Fi exposure (200–500 μW/m²) during 9 days of chicken embryo incubation and assessed the mesonephros at day 9. The authors report no adverse effects on general mesonephros development, but describe moderate degenerative changes and vascular congestion without inflammatory infiltrate. They also report significantly increased apoptotic and proliferating cells and up-regulation of caspase‑1 gene expression, interpreted as disruption of regulatory processes during development.