{"id":"HHS-OASH-2026-0397-0724","body_sha256":"fa5ce59a5c281efb7af3bc73e09145b0bd616a818f20d2213cee28a9c3603db2","status":"ai_reviewed","primary_topic":"research","topics":[{"id":"research","evidence":"-life electromagnetic field exposure and neurodevelopment: a pathophysiological hypothesis and a prospective cohort protocol.\n\nPerspective\nPhysicians and researchers (healthcare professional"},{"id":"children","evidence":"le dosimetry, with objective verification of adherence to nighttime shielding.\n\nChildren\nA differentiated assessment is needed. The 6-18 month window is critical for the maturation of the "},{"id":"neurological","evidence":"sponse to RFI HHS-OASH-2026-0397. Early-life electromagnetic field exposure and neurodevelopment: a pathophysiological hypothesis and a prospective cohort protocol.\n\nPerspective\nPhysicians and res"},{"id":"mechanisms","evidence":" mirror neuron system and grey matter. During this phase, plasticity depends on calcium-dependent mechanisms and on the temporal precision of neuronal activity.\n\nEvidence below current li"},{"id":"template","evidence":"s affiliated with the R&D Department of S.I.S.T.E.M.I. srl [field of activity]. [Funding: specify or declare none.]\n\nReferences\n[1] Garoli A, Greco A. Alpha Psychiatry 2026;27(4):53282. htt"}],"summary":"Proposes an early-life RF exposure and neurodevelopment research protocol, including objective dosimetry and candidate cellular pathways. Explicitly presents a hypothesis rather than human exposure-effect findings; funding and disclosure fields remain unfilled.","reviewed_at":"2026-09-24T19:30:18Z","review_method":"Source-matched AI review of complete published comment bodies; exact supporting passages; attachments not reviewed.","review_source_sha256":"176de8e65ac5432277d2a036d43d45ddcdd444cc3b704d501bdbeb91f185d9ac","source_url":"https://rfsafe.net/hhs-comments/data/catalog.json","scope":"Original comment body; attachments excluded"}